Erythromelanosis follicularis faciei et colli | |
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Specialty | Dermatology |
Erythromelanosis follicularis faciei et colli is an erythematous pigmentary disease involving the follicles, characterized by a reddish-brown, sharply demarcated, symmetrical discoloration involving the preauricular and maxillary regions. [1]: 775–6
Erythromelanosis follicularis faciei et colli is characterized by patches of erythema (with or without telangiectasia), follicular papules (follicular plugging), and bilateral and symmetrical hyperpigmentation (reddish-brown pigmentation) that start on the preauricular areas and cheeks and can eventually migrate to the submandibular portions of the neck. [2]
Although the cause of this disorder is unknown, familial cases, spontaneous mutation, and an autosomal recessive pattern of inheritance have all been documented. [3] [4]
Histopathologic observations include vascular dilatation in the upper dermis, hyperkeratosis, follicular plugging, and enhanced basal membrane pigmentation. [5]
Topical therapies include metronidazole, ammonium lactate, tacalcitol ointment, retinoids, hydroquinone, and salicylic acid. [5] There have been reports of erythema and hyperpigmentation treated with pulsed dye laser or Nd:YAG laser. [6]
Erythromelanosis follicularis faciei et colli | |
---|---|
Specialty | Dermatology |
Erythromelanosis follicularis faciei et colli is an erythematous pigmentary disease involving the follicles, characterized by a reddish-brown, sharply demarcated, symmetrical discoloration involving the preauricular and maxillary regions. [1]: 775–6
Erythromelanosis follicularis faciei et colli is characterized by patches of erythema (with or without telangiectasia), follicular papules (follicular plugging), and bilateral and symmetrical hyperpigmentation (reddish-brown pigmentation) that start on the preauricular areas and cheeks and can eventually migrate to the submandibular portions of the neck. [2]
Although the cause of this disorder is unknown, familial cases, spontaneous mutation, and an autosomal recessive pattern of inheritance have all been documented. [3] [4]
Histopathologic observations include vascular dilatation in the upper dermis, hyperkeratosis, follicular plugging, and enhanced basal membrane pigmentation. [5]
Topical therapies include metronidazole, ammonium lactate, tacalcitol ointment, retinoids, hydroquinone, and salicylic acid. [5] There have been reports of erythema and hyperpigmentation treated with pulsed dye laser or Nd:YAG laser. [6]