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Would
atherosclerosis qualify as a vasculitis ? There is a lot of inflammation, too.
PFHLai 13:13, 2004 May 20 (UTC)
Frankly, Dr. Wolff, I don't know much about Atherosclerosis or Vasculitis. I was hoping to find out their differences from this page on Vasculitis, or the one on Atherosclerosis. Based on what I have read so far, I am under the impression that:
Am I correct ? Should atherosclerosis be considered a special case of slow-progressing vasculitis that is induced/accelerated by fatty deposits in the vessel wall ? Can vasculitic lesions in at a young age become or induce atherosclerotic lesions later in life ? I am looking forward to reading more on vasculitis as your page on this disease grows. Thank you.
PFHLai 03:36, 2004 May 21 (UTC)
Can anyone think of anyone other than Janet Leigh who have died from any type of Vasculitis? -- 198.164.135.37 12:42, 4 Oct 2004 (UTC)
My Child Ryvor Nathon Loyd Anderson May 25 1996 - Dec 3 2006 Passed away At Childrens Hospital in Seattle Washington, due to a condition called Central nervous System Vasculitis of Childhood. The Center for Disease Control, Was the Authority in Conjunction with the King County Medical Examiner. Why the need for names? I found out my son is one of less than 25 in the world. This disease is only found through Autopsy or Brain Biopsy. His symptoms throughout life mimicked And included Cerebral Palsy, Headaches, No weight gain, muscle aches, and finally personality change. His IQ on RECORD is in math 2Xs national average in 2006. No intelligence change was apparent. My child exhibited a early learner attitude born early, and to a prednisone dependent asthmatic mother; born approximately 33 weeks due to increased heartrate. The end result is He appeared to have a stomach flu, and never appeared to regain full consciousness, however motor control allowed for moving him down the hall, an hour later my child became rigid, and was rushed to ER in Everett WA, Transferred To Childrens at Sand Point or Seattle WA. I witnessed swelling then of his eyes, remaining fixed, I held him as they unplugged the Ventilator, and as in birth delivered him into the hands of God, so to speak. I see him often. And know without a doubt one life is missed. That's why I am giving you, a name of who passed into life after death. DA_Anderson@ymail.com have a good day —Preceding unsigned comment added by AngeliaBettina ( talk • contribs) 21:33, 11 October 2010 (UTC)
This is my first edit and I hope my addition of an "external links" section is appropriate. I thought there was a need for a link to the Johns Hopkins page because it contains a lot of valuable information. -- Cameleopard 17:19, 18 July 2005 (UTC)
Atherosclerosis = Vasculitis (FINAL STAGE)
Atherosclerosis is characterized by a non-specific local inflammatory process which is accompanied by a systemic response. A number of prospective studies in initially healthy subjects and in patients with known atherosclerosis have convincingly demonstrated a strong and independent association between even slightly elevated concentrations of various systemic markers of inflammation and a number of cardiovascular events. C-reactive protein (CRP), the classical acute phase protein, seems to be the marker of choice in this clinical situation. The measurement procedure is well standardized and automated, high-sensitivity assays with sufficient precision are available. In addition, recent research has suggested that CRP may not only be a risk marker, but may be directly involved in the pathogenesis of atherosclerosis. Various lifestyle changes and pharmacological interventions are able to reduce elevated CRP concentrations. Testing the inflammation hypothesis should be of major importance in clinical research of atherosclerosis.
Deutsches Ärzteblatt 100, Ausgabe 3 vom 17.01.2003, Seite A-117 / B-108 / C-105 MEDIZIN (Germany)
G.S., MD (Germany)
OK.
We discuss this under the term (unter dem Begriff)
e.g. "DIV" = "Drug-induced Vasculitis" etc.: "DIV is a relatively common cause of inflammatory vasculitis. Drugs from almost every pharmacologic class have been implicated in causing vasculitis in sporadic cases. The level of certainty and quality of evidence for these associations between specific agents and vasculitis vary greatly. The clinical manifestations of DIV range from single organ involvement (most commonly, skin) to life-threatening multiorgan disease." Merkel PA / Rheum Dis Clin North Am. 2001 Nov;27(4):849-62.
or
"TOS" / Toxic-Oil-Syndrome: Spain 1981: Histopathological = a systematic, non necrotizing vasculitis..
or
TILT = Toxicant-induced Loss of Tolerance (Miller & Ashford 1996/98),
TILT-Syndrome & Tilt-Vasculitis:
"The clinical syndromes produced by pollutants are myriad. Some are named and others are not. Discussed separately are small vessel vasculitis, large vessel vasculitis, carotid spasm, Raynaud’s disease, hypertension, specific systemic undifferentiated Tilt-hypersensitive vasculitis, Panarteriitis nodosa, Wegener’s granulomatosis, rheumatoid vasculitis, recurrent phlebitis and thrombophlebitis, thrombosis etc."
"Many necrotizing and non-necrotizing vasculitides are directly caused or closely associated with immunopathogenic mechanism and environmental triggers. It was originally thought that the mechanism most commonly associated with Vasculitis is that of the deposition of circulating immune complexes in the blood vessel walls. However, this association appears to be limited since com-plexes are not always necessary for inflammation of vessels to occur. In fact, a larger percentage of blood vessels deregulation in the hypersensitive (pseudoallergic, intolerant) patient appears to occur through non-immune mechanism." ...
G.S., MD, Germany EU
With respect to the "Cultural References" section of the article, my friend with vasculitis told me that the reason Dr. House refers to it a lot is because a vasculitis society paid NBC to raise awareness about the disease.
The symptoms of Polyarteritis Nodosa listed on this page don't really match the symptoms described in the main article on Polyarteritis Nodosa... I think the diagnositc criteria listed here are probably more accurate, maybe this needs adding to the other page?
![]() | This article may be too technical for most readers to understand.(September 2010) |
this really really needs to be made more accessible. it reads like something out of medical school rather than a standard wikipedia article. I wouldn't doubt the information as it certainly does sound authoritative and correct when presented like this... however, I don't think most people will really be able to understand it! it's got way too much medical jargon and the writing style is choppy.
According to this Chicago Tribune article, http://www.chicagotribune.com/entertainment/chi-harold-ramis-dead-20140224,0,2259309.story , Mr Ramis died of "autoimmune inflammatory vasculitis". Is this one of the vasculitis conditions covered by the article, though I don't find the term there. In fact, no "autoimmune" anything is mentioned. As people become aware of his death, more will probably browser-search the term and expect WP to address it. Is there another article that covers it that I cannot find by searching WP? Thank you, Wordreader ( talk) 19:05, 24 February 2014 (UTC)
I wanted to discuss this before I make any major edits as this is a very popular page. I recently had a discussion on the medicine wikiproject regarding better classifications of the different types of vasculitis. I propose that we make a seprate section for classification as it's more relevant to this topic. I've also reformatted the classification to fit the most recent guidelines. My sandbox can be viewed here if anyone has any feedback. CursedWithTheAbilityToDoTheMath ( talk) 17:42, 14 January 2024 (UTC)
![]() | This article is rated C-class on Wikipedia's
content assessment scale. It is of interest to the following WikiProjects: | ||||||||||||||||||||||
|
![]() | Ideal sources for Wikipedia's health content are defined in the guideline
Wikipedia:Identifying reliable sources (medicine) and are typically
review articles. Here are links to possibly useful sources of information about Vasculitis.
|
Would
atherosclerosis qualify as a vasculitis ? There is a lot of inflammation, too.
PFHLai 13:13, 2004 May 20 (UTC)
Frankly, Dr. Wolff, I don't know much about Atherosclerosis or Vasculitis. I was hoping to find out their differences from this page on Vasculitis, or the one on Atherosclerosis. Based on what I have read so far, I am under the impression that:
Am I correct ? Should atherosclerosis be considered a special case of slow-progressing vasculitis that is induced/accelerated by fatty deposits in the vessel wall ? Can vasculitic lesions in at a young age become or induce atherosclerotic lesions later in life ? I am looking forward to reading more on vasculitis as your page on this disease grows. Thank you.
PFHLai 03:36, 2004 May 21 (UTC)
Can anyone think of anyone other than Janet Leigh who have died from any type of Vasculitis? -- 198.164.135.37 12:42, 4 Oct 2004 (UTC)
My Child Ryvor Nathon Loyd Anderson May 25 1996 - Dec 3 2006 Passed away At Childrens Hospital in Seattle Washington, due to a condition called Central nervous System Vasculitis of Childhood. The Center for Disease Control, Was the Authority in Conjunction with the King County Medical Examiner. Why the need for names? I found out my son is one of less than 25 in the world. This disease is only found through Autopsy or Brain Biopsy. His symptoms throughout life mimicked And included Cerebral Palsy, Headaches, No weight gain, muscle aches, and finally personality change. His IQ on RECORD is in math 2Xs national average in 2006. No intelligence change was apparent. My child exhibited a early learner attitude born early, and to a prednisone dependent asthmatic mother; born approximately 33 weeks due to increased heartrate. The end result is He appeared to have a stomach flu, and never appeared to regain full consciousness, however motor control allowed for moving him down the hall, an hour later my child became rigid, and was rushed to ER in Everett WA, Transferred To Childrens at Sand Point or Seattle WA. I witnessed swelling then of his eyes, remaining fixed, I held him as they unplugged the Ventilator, and as in birth delivered him into the hands of God, so to speak. I see him often. And know without a doubt one life is missed. That's why I am giving you, a name of who passed into life after death. DA_Anderson@ymail.com have a good day —Preceding unsigned comment added by AngeliaBettina ( talk • contribs) 21:33, 11 October 2010 (UTC)
This is my first edit and I hope my addition of an "external links" section is appropriate. I thought there was a need for a link to the Johns Hopkins page because it contains a lot of valuable information. -- Cameleopard 17:19, 18 July 2005 (UTC)
Atherosclerosis = Vasculitis (FINAL STAGE)
Atherosclerosis is characterized by a non-specific local inflammatory process which is accompanied by a systemic response. A number of prospective studies in initially healthy subjects and in patients with known atherosclerosis have convincingly demonstrated a strong and independent association between even slightly elevated concentrations of various systemic markers of inflammation and a number of cardiovascular events. C-reactive protein (CRP), the classical acute phase protein, seems to be the marker of choice in this clinical situation. The measurement procedure is well standardized and automated, high-sensitivity assays with sufficient precision are available. In addition, recent research has suggested that CRP may not only be a risk marker, but may be directly involved in the pathogenesis of atherosclerosis. Various lifestyle changes and pharmacological interventions are able to reduce elevated CRP concentrations. Testing the inflammation hypothesis should be of major importance in clinical research of atherosclerosis.
Deutsches Ärzteblatt 100, Ausgabe 3 vom 17.01.2003, Seite A-117 / B-108 / C-105 MEDIZIN (Germany)
G.S., MD (Germany)
OK.
We discuss this under the term (unter dem Begriff)
e.g. "DIV" = "Drug-induced Vasculitis" etc.: "DIV is a relatively common cause of inflammatory vasculitis. Drugs from almost every pharmacologic class have been implicated in causing vasculitis in sporadic cases. The level of certainty and quality of evidence for these associations between specific agents and vasculitis vary greatly. The clinical manifestations of DIV range from single organ involvement (most commonly, skin) to life-threatening multiorgan disease." Merkel PA / Rheum Dis Clin North Am. 2001 Nov;27(4):849-62.
or
"TOS" / Toxic-Oil-Syndrome: Spain 1981: Histopathological = a systematic, non necrotizing vasculitis..
or
TILT = Toxicant-induced Loss of Tolerance (Miller & Ashford 1996/98),
TILT-Syndrome & Tilt-Vasculitis:
"The clinical syndromes produced by pollutants are myriad. Some are named and others are not. Discussed separately are small vessel vasculitis, large vessel vasculitis, carotid spasm, Raynaud’s disease, hypertension, specific systemic undifferentiated Tilt-hypersensitive vasculitis, Panarteriitis nodosa, Wegener’s granulomatosis, rheumatoid vasculitis, recurrent phlebitis and thrombophlebitis, thrombosis etc."
"Many necrotizing and non-necrotizing vasculitides are directly caused or closely associated with immunopathogenic mechanism and environmental triggers. It was originally thought that the mechanism most commonly associated with Vasculitis is that of the deposition of circulating immune complexes in the blood vessel walls. However, this association appears to be limited since com-plexes are not always necessary for inflammation of vessels to occur. In fact, a larger percentage of blood vessels deregulation in the hypersensitive (pseudoallergic, intolerant) patient appears to occur through non-immune mechanism." ...
G.S., MD, Germany EU
With respect to the "Cultural References" section of the article, my friend with vasculitis told me that the reason Dr. House refers to it a lot is because a vasculitis society paid NBC to raise awareness about the disease.
The symptoms of Polyarteritis Nodosa listed on this page don't really match the symptoms described in the main article on Polyarteritis Nodosa... I think the diagnositc criteria listed here are probably more accurate, maybe this needs adding to the other page?
![]() | This article may be too technical for most readers to understand.(September 2010) |
this really really needs to be made more accessible. it reads like something out of medical school rather than a standard wikipedia article. I wouldn't doubt the information as it certainly does sound authoritative and correct when presented like this... however, I don't think most people will really be able to understand it! it's got way too much medical jargon and the writing style is choppy.
According to this Chicago Tribune article, http://www.chicagotribune.com/entertainment/chi-harold-ramis-dead-20140224,0,2259309.story , Mr Ramis died of "autoimmune inflammatory vasculitis". Is this one of the vasculitis conditions covered by the article, though I don't find the term there. In fact, no "autoimmune" anything is mentioned. As people become aware of his death, more will probably browser-search the term and expect WP to address it. Is there another article that covers it that I cannot find by searching WP? Thank you, Wordreader ( talk) 19:05, 24 February 2014 (UTC)
I wanted to discuss this before I make any major edits as this is a very popular page. I recently had a discussion on the medicine wikiproject regarding better classifications of the different types of vasculitis. I propose that we make a seprate section for classification as it's more relevant to this topic. I've also reformatted the classification to fit the most recent guidelines. My sandbox can be viewed here if anyone has any feedback. CursedWithTheAbilityToDoTheMath ( talk) 17:42, 14 January 2024 (UTC)