The following
outline is provided as an overview of and topical guide to diabetes mellitus (diabetes insipidus not included below):
Diabetes mellitus – group of metabolic diseases in which a person has high
blood sugar, either because the
pancreas does not produce enough
insulin, or because cells do not respond properly to the insulin that is produced,[1] a condition called
insulin resistance. The resultant high blood sugar produces the classical symptoms of
polyuria (frequent urination),
polydipsia (increased thirst) and
polyphagia (increased hunger).
Type 1 diabetes – disease that results in autoimmune destruction of insulin-producing beta cells of the pancreas.[4]
Type 2 diabetes – metabolic disorder that is characterized by high blood glucose in the context of insulin resistance and relative insulin deficiency.[5]
Disease of affluence – type 2 diabetes is one of the "diseases of affluence", which include mostly chronic non-communicable diseases for which personal lifestyles and societal conditions associated with economic development are believed to be important risk factors.
Gestational diabetes – Gestational diabetes, is a temporary condition that is first diagnosed during pregnancy. Like type 1 and type 2 diabetes, gestational diabetes causes blood sugar levels to become too high. It involves an increased risk of developing diabetes for both mother and child.
Symptoms of prediabetes – prediabetes typically has no distinct signs or symptoms. Patients should monitor for signs and symptoms of type 2 diabetes mellitus (see below).
Regular
physical exercise – in addition to helping to maintain a healthy weight, sufficient vigorous physical exercise increases cells' sensitivity to insulin, and can thus prevent and possibly revert insulin resistance.
Medication – specific medications have been shown to be able to prevent type 2 diabetes. However, the disease can often be delayed through proper nutrition and regular exercise.[7]
^Kumar, Vinay; Fausto, Nelson; Abbas, Abul K.; Cotran, Ramzi S.; Robbins, Stanley L. (2005). Robbins and Cotran Pathologic Basis of Disease (7th ed.). Philadelphia, Pa.: Saunders. pp. 1194–1195.
ISBN0-7216-0187-1.
The following
outline is provided as an overview of and topical guide to diabetes mellitus (diabetes insipidus not included below):
Diabetes mellitus – group of metabolic diseases in which a person has high
blood sugar, either because the
pancreas does not produce enough
insulin, or because cells do not respond properly to the insulin that is produced,[1] a condition called
insulin resistance. The resultant high blood sugar produces the classical symptoms of
polyuria (frequent urination),
polydipsia (increased thirst) and
polyphagia (increased hunger).
Type 1 diabetes – disease that results in autoimmune destruction of insulin-producing beta cells of the pancreas.[4]
Type 2 diabetes – metabolic disorder that is characterized by high blood glucose in the context of insulin resistance and relative insulin deficiency.[5]
Disease of affluence – type 2 diabetes is one of the "diseases of affluence", which include mostly chronic non-communicable diseases for which personal lifestyles and societal conditions associated with economic development are believed to be important risk factors.
Gestational diabetes – Gestational diabetes, is a temporary condition that is first diagnosed during pregnancy. Like type 1 and type 2 diabetes, gestational diabetes causes blood sugar levels to become too high. It involves an increased risk of developing diabetes for both mother and child.
Symptoms of prediabetes – prediabetes typically has no distinct signs or symptoms. Patients should monitor for signs and symptoms of type 2 diabetes mellitus (see below).
Regular
physical exercise – in addition to helping to maintain a healthy weight, sufficient vigorous physical exercise increases cells' sensitivity to insulin, and can thus prevent and possibly revert insulin resistance.
Medication – specific medications have been shown to be able to prevent type 2 diabetes. However, the disease can often be delayed through proper nutrition and regular exercise.[7]
^Kumar, Vinay; Fausto, Nelson; Abbas, Abul K.; Cotran, Ramzi S.; Robbins, Stanley L. (2005). Robbins and Cotran Pathologic Basis of Disease (7th ed.). Philadelphia, Pa.: Saunders. pp. 1194–1195.
ISBN0-7216-0187-1.