This user is a student editor in
Wikipedia:Wiki_Ed/Carleton_College/Immunology_(Winter). Student assignments should always be carried out using a course page set up by the instructor. It is usually best to develop assignments in your sandbox. After evaluation, the additions may go on to become a Wikipedia article or be published in an existing article. |
Student enrolled in Carleton College Biology 310: Immunology.
For my main article project/Assignment #4, I will be adding to the page on Neonatal Fc receptor.
I want to add cited information about the role of FcRn and where it is expressed in adults. [1] I want to add an image on the mechanism of maternal IgG transfer to clarify the process. [2] Here is another source that details the role of FcRn in different organs of the body and also the implications of FcRn-IgG binding for immunotherapies. [3] There is already a short section in the current article about drugs that use FcRn to increase their half-lives, but I could add some more information. This next article has some interesting information about FcRn-IgG binding inhibitors as a possible treatment for autoimmune diseases that are caused by self-antigen-attacking IgG antibodies. [4] And finally, there is also plenty of evidence suggesting that FcRn plays a role in extending the half-life of serum albumin and not just IgG. [5]
This user is a student editor in
Wikipedia:Wiki_Ed/Carleton_College/Immunology_(Winter). Student assignments should always be carried out using a course page set up by the instructor. It is usually best to develop assignments in your sandbox. After evaluation, the additions may go on to become a Wikipedia article or be published in an existing article. |
Student enrolled in Carleton College Biology 310: Immunology.
For my main article project/Assignment #4, I will be adding to the page on Neonatal Fc receptor.
I want to add cited information about the role of FcRn and where it is expressed in adults. [1] I want to add an image on the mechanism of maternal IgG transfer to clarify the process. [2] Here is another source that details the role of FcRn in different organs of the body and also the implications of FcRn-IgG binding for immunotherapies. [3] There is already a short section in the current article about drugs that use FcRn to increase their half-lives, but I could add some more information. This next article has some interesting information about FcRn-IgG binding inhibitors as a possible treatment for autoimmune diseases that are caused by self-antigen-attacking IgG antibodies. [4] And finally, there is also plenty of evidence suggesting that FcRn plays a role in extending the half-life of serum albumin and not just IgG. [5]