From Wikipedia, the free encyclopedia
  • Comment: This is at present a duplicate of the article on electromagnetic hypersensitivity. There is also a sourcing issue here, in that Wikipedia prefers secondary and tertiary sources, as primary sources so often lack context. This is particularly important for contentious topics, where the many primary research papers often give conflicting findings. Most of the papers cited are primary and risk the accusation of cherry-picking in an attempt to support an untenable thesis. They can be used to support the findings of reliable and peer-reviewed secondary and tertiary sources (overviews, popular journal articles, books, etc.) but not to verify any particular thesis. Another issue is the need to cite inline the source of any given contentious claim and not leave the reader to wonder where in the bibliography it might be found. Some useful explanations of all this may be fond at WP:RS, WP:CITE and, in the present context, WP:MEDRS. It is particularly important that you highlight any secondary sources, as defined at WP:MEDDEF, that you have cited, and/or find additional ones to cite. I would also suggest that you format this draft as a set of discrete topics or passages which might be added to the EHS or MCS articles or possibly elsewhere, as it becomes much harder then for the nay-sayers to point at breaches of procedure. All this comes with no promise from me that Wikipedia's current take on the science may not prevail, I remain open on this matter. But I will try to stop procedural wrangling from getting in the way of the science. — Cheers, Steelpillow ( Talk) 11:00, 13 December 2016 (UTC)


==Notes on Electromagnetic Hypersensitivity (EHS) ISB22 ( talk) 18:36, 15 December 2016 (UTC)

Edits to current entry:

Some scientists have been able to conduct successful provocation studies, however, showing that "thyroid dysfunction, liver dysfunction and chronic inflammatory processes in small but remarkable fractions of EHS sufferers as potential sources of symptoms that merit further investigation in future studies. In the cases of TSH and ALT/AST there were significant differences between cases and controls. The hypotheses of anaemia or kidney dysfunction playing a major role in EHS could be unambiguously refuted. Clinically it is recommended to check for signs of treatable somatic conditions when caring for individuals suffering from self-proclaimed EHS. "Our results identified laboratory signs of thyroid dysfunction, liver dysfunction and chronic inflammatory processes in small but remarkable fractions of EHS sufferers as potential sources of symptoms that merit further investigation in future studies" [1]

Professor Dominique Belpomme found changes in blood molecules (heat shock proteins) and histamine levels that were markedly altered in a highly significant proportion of those tested, in comparison to the control group. Additionally a reduced circulation of blood to the brain compared to the control group was found. They concluded that EHS is “a real physical illness, like cancer or heart disease”. [2]

Professor Magda Havas, a Trent University environmental scientist, used a wireless computer and a router on an EHS adult male. In a blind test Havas exposed him to microwave radiation at levels similar to those in an average wireless classroom. She found the closer the router, the faster the subject’s heart rate. Although the levels she used in the test were under Health Canada’s limit, Havas has a theory that may explain her findings. “Some percentage of the population is reacting to this microwave radiation” [3]

References

  1. ^ Bioelectromagnetics. 2009 May;30(4):299-306. doi: 10.1002/bem.20486. Blood laboratory findings in patients suffering from self-perceived electromagnetic hypersensitivity (EHS). Dahmen N1, Ghezel-Ahmadi D, Engel A.
  2. ^ Belpomme et al: “Reliable disease biomarkers characterising and identifying Electrohypersensitivity and Multiple Chemical Sensitivity as two etiopathogenic aspect of a unique pathological disorder.” Rev Environ Health, 2015. PMID  266133326, pdf
  3. ^ Rev Environ Health. 2013;28(2-3):75-84. doi: 10.1515/reveh-2013-0004. Radiation from wireless technology affects the blood, the heart, and the autonomic nervous system. Havas M.

These studies show similar results

International Journal of Neuroscience: ELECTROMAGNETIC HYPERSENSITIVITY: EVIDENCE FOR A NOVEL NEUROLOGICAL SYNDROME David E. McCarty, M.D., Simona Carrubba, Ph.D., Andrew L. Chesson, Jr., M.D., Clifton Frilot, I, Ph.D., Eduardo Gonzalez-Toledo, M.D., Andrew A. Marino, Ph.D. doi:10.3109/00207454.2011.608139

Electromagn Biol Med. 2013 Sep;32(3):281-90. doi: 10.3109/15368378.2012.712586. Epub 2013 Jan 9. Hypothesis on how to measure electromagnetic hypersensitivity. Tuengler A1, von Klitzing L.

Metabolic and Genetic Screening of Electromagnetic Hypersensitive Subjects as a Feasible Tool for Diagnostics and Intervention, Mediators of Inflammation Volume 2014 (2014), Article ID 924184, 14 pages http://dx.doi.org/10.1155/2014/924184 Chiara De Luca, Jeffrey Chung Sheun Thai, Desanka Raskovic, Eleonora Cesareo, Daniela Caccamo, Arseny Trukhanov, and Liudmila Korkina1 <ref>

From Wikipedia, the free encyclopedia
  • Comment: This is at present a duplicate of the article on electromagnetic hypersensitivity. There is also a sourcing issue here, in that Wikipedia prefers secondary and tertiary sources, as primary sources so often lack context. This is particularly important for contentious topics, where the many primary research papers often give conflicting findings. Most of the papers cited are primary and risk the accusation of cherry-picking in an attempt to support an untenable thesis. They can be used to support the findings of reliable and peer-reviewed secondary and tertiary sources (overviews, popular journal articles, books, etc.) but not to verify any particular thesis. Another issue is the need to cite inline the source of any given contentious claim and not leave the reader to wonder where in the bibliography it might be found. Some useful explanations of all this may be fond at WP:RS, WP:CITE and, in the present context, WP:MEDRS. It is particularly important that you highlight any secondary sources, as defined at WP:MEDDEF, that you have cited, and/or find additional ones to cite. I would also suggest that you format this draft as a set of discrete topics or passages which might be added to the EHS or MCS articles or possibly elsewhere, as it becomes much harder then for the nay-sayers to point at breaches of procedure. All this comes with no promise from me that Wikipedia's current take on the science may not prevail, I remain open on this matter. But I will try to stop procedural wrangling from getting in the way of the science. — Cheers, Steelpillow ( Talk) 11:00, 13 December 2016 (UTC)


==Notes on Electromagnetic Hypersensitivity (EHS) ISB22 ( talk) 18:36, 15 December 2016 (UTC)

Edits to current entry:

Some scientists have been able to conduct successful provocation studies, however, showing that "thyroid dysfunction, liver dysfunction and chronic inflammatory processes in small but remarkable fractions of EHS sufferers as potential sources of symptoms that merit further investigation in future studies. In the cases of TSH and ALT/AST there were significant differences between cases and controls. The hypotheses of anaemia or kidney dysfunction playing a major role in EHS could be unambiguously refuted. Clinically it is recommended to check for signs of treatable somatic conditions when caring for individuals suffering from self-proclaimed EHS. "Our results identified laboratory signs of thyroid dysfunction, liver dysfunction and chronic inflammatory processes in small but remarkable fractions of EHS sufferers as potential sources of symptoms that merit further investigation in future studies" [1]

Professor Dominique Belpomme found changes in blood molecules (heat shock proteins) and histamine levels that were markedly altered in a highly significant proportion of those tested, in comparison to the control group. Additionally a reduced circulation of blood to the brain compared to the control group was found. They concluded that EHS is “a real physical illness, like cancer or heart disease”. [2]

Professor Magda Havas, a Trent University environmental scientist, used a wireless computer and a router on an EHS adult male. In a blind test Havas exposed him to microwave radiation at levels similar to those in an average wireless classroom. She found the closer the router, the faster the subject’s heart rate. Although the levels she used in the test were under Health Canada’s limit, Havas has a theory that may explain her findings. “Some percentage of the population is reacting to this microwave radiation” [3]

References

  1. ^ Bioelectromagnetics. 2009 May;30(4):299-306. doi: 10.1002/bem.20486. Blood laboratory findings in patients suffering from self-perceived electromagnetic hypersensitivity (EHS). Dahmen N1, Ghezel-Ahmadi D, Engel A.
  2. ^ Belpomme et al: “Reliable disease biomarkers characterising and identifying Electrohypersensitivity and Multiple Chemical Sensitivity as two etiopathogenic aspect of a unique pathological disorder.” Rev Environ Health, 2015. PMID  266133326, pdf
  3. ^ Rev Environ Health. 2013;28(2-3):75-84. doi: 10.1515/reveh-2013-0004. Radiation from wireless technology affects the blood, the heart, and the autonomic nervous system. Havas M.

These studies show similar results

International Journal of Neuroscience: ELECTROMAGNETIC HYPERSENSITIVITY: EVIDENCE FOR A NOVEL NEUROLOGICAL SYNDROME David E. McCarty, M.D., Simona Carrubba, Ph.D., Andrew L. Chesson, Jr., M.D., Clifton Frilot, I, Ph.D., Eduardo Gonzalez-Toledo, M.D., Andrew A. Marino, Ph.D. doi:10.3109/00207454.2011.608139

Electromagn Biol Med. 2013 Sep;32(3):281-90. doi: 10.3109/15368378.2012.712586. Epub 2013 Jan 9. Hypothesis on how to measure electromagnetic hypersensitivity. Tuengler A1, von Klitzing L.

Metabolic and Genetic Screening of Electromagnetic Hypersensitive Subjects as a Feasible Tool for Diagnostics and Intervention, Mediators of Inflammation Volume 2014 (2014), Article ID 924184, 14 pages http://dx.doi.org/10.1155/2014/924184 Chiara De Luca, Jeffrey Chung Sheun Thai, Desanka Raskovic, Eleonora Cesareo, Daniela Caccamo, Arseny Trukhanov, and Liudmila Korkina1 <ref>


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