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If anyone has a photograph (fair use) of the cherry red spot, that would be great! Rytyho usa ( talk) 03:06, 2 February 2014 (UTC)
Hello, we are a group of medical student’s from Queen’s University. We are working to improve this article over the next month and will posting our planned changes on this talk page. We look forward to working with the existing Wikipedia medical editing community to improve this article and share evidence. We welcome feedback and suggestions as we learn to edit. Thank you. 14tcm1 ( talk) 20:19, 11 November 2019 (UTC)
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References
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help)CS1 maint: PMC format (
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Proposed change for treatment:
Several treatments have been attempted for CRAO, however, none show definitive improvement in outcomes. Current therapeutic modalities of CRAO include the following: citation needed
To achieve the best outcome for the person with CRAO, it is primordial to identify the condition in a timely manner and to refer to the appropriate specialist. [1]
References
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cite journal}}
: Check |doi=
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help)
18j03 ( talk) 01:18, 19 November 2019 (UTC)
Proposed Changes for the Mechanism section: 1) Remove reference used in existing sentences, and use this reference instead. [1] Rationale for proposed change: The citation does not lead to a reference, the new reference states the same understanding of the central retinal artery anatomy and course. There may be disagreement whether to keep the same material with a new reference however this is widely accepted anatomy and therefore I believe it is acceptable to just provide the new reliable reference.
2) Additional sentences to add following the last sentence of "Mechanism" section: Central retinal artery occlusion is most often due to emboli blocking the artery and therefore prevents the artery from delivering nutrients to most of the retina. These emboli originate from the carotid arteries most of the time but in 25% of cases, this is due to plaque build-up in the ophthalmic artery. The most frequent site of blockage is at the most narrow part of the artery which is where the artery pierces the dura covering the optic nerve. [2]. Rationale for proposed change: Though other sections of the article contribute some insight to understanding this component of central retinal artery occlusion, a gap in information was noticed between the discussion of the artery's anatomy and the causes of its occlusion by emboli. These sentences would serve to connect these discussions and provide a more complete picture. There may be variation in the opinion of the frequency that emboli originate within the artery itself but the stated 25% rate will be supported with reliable evidence.
References
Oginty ( talk • contribs) 02:24, 19 November 2019 (UTC)
18mlk7 ( talk) 02:47, 19 November 2019 (UTC) Proposed changes for signs and symptoms section: "Central retinal artery occlusion is characterized by painless, acute vision loss in one eye. [1] Upon fundoscopic exam, one would expect to find: cherry-red spot (Figure X) (90%), retinal opacity in the posterior pole (Figure Y) (58%), pallor (39%), retinal arterial attenuation (32%), and optic disk edema (22%). [1] During later stages of onset, one may also find plaques, emboli, and optic atrophy. [1]"
This section will include an example figure for the cherry-red spot and retinal opacity in the posterior pole (the two most commonly seen signs) were included due to their prevalence. The retinal opacity of the posterior pole was also not able to be hyperlinked to an existing page for clarity (as it is a more visual phenomenon) thus an image would be the most effective way to explain this sign.
References
Proposed changes for Causes section:
"Causes of CRAO may be examined based on its various classifications, including non-arteritic and arteritic CRAO. [1] [2] [3] Non-arteritic CRAO is most commonly caused by an embolus and occlusion at the narrowest part of the carotid retinal artery due to plaques in the carotid artery resulting in carotid retinal artery atherosclerosis. [1] [2] [3] Further causes of non-arteritic CRAO may include vasculitis and chronic systemic autoimmune diseases. [1] Arteritic CRAO is most commonly caused by giant cell arteritis." [1] [3]
Rationale for proposed changes: First sentence: the aim of this sentence was to introduce readers to the existence of and distinction between various subclassifications of CRAO, and how different classifications have different causes. The previous version of the article did not introduce such classifications, and was not specific as to which type of CRAO can be attributable to which cause. The organization of CRAO into subclassifications was consistent across all three cited sources, therefore I believe its inclusion is justified. Second sentence: while the aim of the original sentence of how carotid artery atherosclerosis is a common cause of CRAO was maintained, this sentence was expanded on to explain how atherosclerosis can lead to the formation of an embolus in non-arteritic CRAO specifically, which was brought up by all three cited authors. Third sentence: this sentence was added to include additional causes of non-arteritic CRAO other than embolus. These additional causes are supported by both Dr. Sanjay Sharma and Dr. Sohan Hayreh, a leading expert in this field. Fourth sentence: this sentence was modified to remove the age of 70 years old as a risk factor for giant cell arteritis, as information regarding which age group this complication was most prevalent in varied in the literature, and no one age was found to be definite. The sentence was restructured to emphasize the main point of giant cell arteritis being the main cause of arteritic CRAO, as evidenced by consistent information across all three sources.
{{
cite journal}}
: CS1 maint: unflagged free DOI (
link)
This article is rated Start-class on Wikipedia's
content assessment scale. It is of interest to the following WikiProjects: | |||||||||||||||||
|
It is requested that a photograph be
included in this article to
improve its quality.
The external tool WordPress Openverse may be able to locate suitable images on Flickr and other web sites. |
If anyone has a photograph (fair use) of the cherry red spot, that would be great! Rytyho usa ( talk) 03:06, 2 February 2014 (UTC)
Hello, we are a group of medical student’s from Queen’s University. We are working to improve this article over the next month and will posting our planned changes on this talk page. We look forward to working with the existing Wikipedia medical editing community to improve this article and share evidence. We welcome feedback and suggestions as we learn to edit. Thank you. 14tcm1 ( talk) 20:19, 11 November 2019 (UTC)
{{
cite journal}}
: Check date values in: |date=
(
help)CS1 maint: unflagged free DOI (
link)
{{
cite journal}}
: CS1 maint: unflagged free DOI (
link)
References
{{
cite journal}}
: Check date values in: |date=
(
help)CS1 maint: PMC format (
link) CS1 maint: unflagged free DOI (
link)
{{
cite journal}}
: Check date values in: |date=
(
help)
Proposed change for treatment:
Several treatments have been attempted for CRAO, however, none show definitive improvement in outcomes. Current therapeutic modalities of CRAO include the following: citation needed
To achieve the best outcome for the person with CRAO, it is primordial to identify the condition in a timely manner and to refer to the appropriate specialist. [1]
References
{{
cite journal}}
: Check |doi=
value (
help)
18j03 ( talk) 01:18, 19 November 2019 (UTC)
Proposed Changes for the Mechanism section: 1) Remove reference used in existing sentences, and use this reference instead. [1] Rationale for proposed change: The citation does not lead to a reference, the new reference states the same understanding of the central retinal artery anatomy and course. There may be disagreement whether to keep the same material with a new reference however this is widely accepted anatomy and therefore I believe it is acceptable to just provide the new reliable reference.
2) Additional sentences to add following the last sentence of "Mechanism" section: Central retinal artery occlusion is most often due to emboli blocking the artery and therefore prevents the artery from delivering nutrients to most of the retina. These emboli originate from the carotid arteries most of the time but in 25% of cases, this is due to plaque build-up in the ophthalmic artery. The most frequent site of blockage is at the most narrow part of the artery which is where the artery pierces the dura covering the optic nerve. [2]. Rationale for proposed change: Though other sections of the article contribute some insight to understanding this component of central retinal artery occlusion, a gap in information was noticed between the discussion of the artery's anatomy and the causes of its occlusion by emboli. These sentences would serve to connect these discussions and provide a more complete picture. There may be variation in the opinion of the frequency that emboli originate within the artery itself but the stated 25% rate will be supported with reliable evidence.
References
Oginty ( talk • contribs) 02:24, 19 November 2019 (UTC)
18mlk7 ( talk) 02:47, 19 November 2019 (UTC) Proposed changes for signs and symptoms section: "Central retinal artery occlusion is characterized by painless, acute vision loss in one eye. [1] Upon fundoscopic exam, one would expect to find: cherry-red spot (Figure X) (90%), retinal opacity in the posterior pole (Figure Y) (58%), pallor (39%), retinal arterial attenuation (32%), and optic disk edema (22%). [1] During later stages of onset, one may also find plaques, emboli, and optic atrophy. [1]"
This section will include an example figure for the cherry-red spot and retinal opacity in the posterior pole (the two most commonly seen signs) were included due to their prevalence. The retinal opacity of the posterior pole was also not able to be hyperlinked to an existing page for clarity (as it is a more visual phenomenon) thus an image would be the most effective way to explain this sign.
References
Proposed changes for Causes section:
"Causes of CRAO may be examined based on its various classifications, including non-arteritic and arteritic CRAO. [1] [2] [3] Non-arteritic CRAO is most commonly caused by an embolus and occlusion at the narrowest part of the carotid retinal artery due to plaques in the carotid artery resulting in carotid retinal artery atherosclerosis. [1] [2] [3] Further causes of non-arteritic CRAO may include vasculitis and chronic systemic autoimmune diseases. [1] Arteritic CRAO is most commonly caused by giant cell arteritis." [1] [3]
Rationale for proposed changes: First sentence: the aim of this sentence was to introduce readers to the existence of and distinction between various subclassifications of CRAO, and how different classifications have different causes. The previous version of the article did not introduce such classifications, and was not specific as to which type of CRAO can be attributable to which cause. The organization of CRAO into subclassifications was consistent across all three cited sources, therefore I believe its inclusion is justified. Second sentence: while the aim of the original sentence of how carotid artery atherosclerosis is a common cause of CRAO was maintained, this sentence was expanded on to explain how atherosclerosis can lead to the formation of an embolus in non-arteritic CRAO specifically, which was brought up by all three cited authors. Third sentence: this sentence was added to include additional causes of non-arteritic CRAO other than embolus. These additional causes are supported by both Dr. Sanjay Sharma and Dr. Sohan Hayreh, a leading expert in this field. Fourth sentence: this sentence was modified to remove the age of 70 years old as a risk factor for giant cell arteritis, as information regarding which age group this complication was most prevalent in varied in the literature, and no one age was found to be definite. The sentence was restructured to emphasize the main point of giant cell arteritis being the main cause of arteritic CRAO, as evidenced by consistent information across all three sources.
{{
cite journal}}
: CS1 maint: unflagged free DOI (
link)