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It seems like a tough question whenever one wants to answer precisely (is it more 1% or more 5%) because it depends on the country, the age, and mostly the criterion chosen (one line, two line difference? including all amblyogenic causes or not?). However, when one is claiming that it is between 1 and 5%, ALL studies agree (in western countries). So I don't think so we can go wrong on this one. They agree no matter what is the age or the criterion, but the range can go to 10% when including non-western countries.
Secondary sources:
-- Adrien Chopin ( talk) 07:52, 21 April 2016 (UTC)
It was quite ironic that Amblyopia is one of the very few diseases that is not tamed enough by the human intelligence. It almost goes unnoticed at the very age of the victim, when it can be best controlled. It is good that children are lucky enough to have wonderful treatment regimens available to them these days, if only, when detected in time, that is in their very early years, but the stigma and pain that is associated with Adult Amblyopia is particularly unbearable. I came to know that I am one of the very few people who fell to this seemingly innocuous disease(rather poor vision condition - doctors won't give up to call it a disease) when I was first disqualified in 'Army Selections'. That was a blow to my career as I always dreamt of becoming an 'Officer in Uniform' right from my childhood. Then started my ordeal, it forced me to change my goals, take up unplanned ventures, constantly irritated me now and then, and more than anything else, I am very much depressed to imagine the fact that one day I will become a one-eyed jack.
While it is extremely painful to learn that there is no proven treatment for the adults, the plight of the people like me in the still developing countries is un-imaginable, where nothing is being done effectively, even the diagnosis of this bloody disease( let me vent my anger).
My Dear Brothers Of The World Who Are In The Same Boat With Me: We share the slogan with the victims of the most dreaded disease(or ???condition) on this earth AIDS:
DOCTORS OF THE WORLD: DO SOMETHING AND SAVE US before it is too late.
Does Amblyopia have anything to do with Ocular Dominance?
Ocular Dominance Wikipedia link:
http://en.wikipedia.org/wiki/Ocular_dominance
Amblyopia has a lot to do with ocular dominance, though not necessarily in a clear-cut way. Monocular occlusion during development is a well-known way to cause amblyopia, and causes the ocular dominance columns of cortex for the dominant eye of the amblyope to dominate that of the amblyopic eye. Behaviorally, amblyopia - which has a poor medical definition - exhibits symptoms that suggest information loss in transmission from eye to cortex because of poor development. That information loss could be caused by the mechanics of the screwed up distribution of ocular dominance cells, which extends in a loop from the eye through the lateral geniculate nucleus, and into the cortex, as well as into the pons which controls certain automatic tracking activities.<spetz>. 216.175.188.98 22:17, 28 February 2007 (UTC)
The controversial aspect is covered by the bates method article. It now looks as if the claim of some is controversial. Seeyou 22:03, 18 January 2007 (UTC)
About the deletion lazy eye or tense eye. WIkipedia also has the policy of a neutral point of view. That means when there are substantial bodies of opposing opinions, they all need to be presented. The formula is "A says B about C." You can't say in the article "mainstream medicine is bunk." You can't say in a vague sort of way "Bates supporters think mainstream medicine is bunk." You can say "so-and-so, in his book Thus-and-such, says on page 234, 'mainstream medicine is bunk.'". Seeyou 18:50, 25 January 2007 (UTC)
> Look your vague references to eyes being 'tense' as opposed to 'lazy' or 'poor' suggest that you don't know what amblyopia is. Why is the reference vague, please explain. suggest that you don't know what amblyopia is ? That is your opinion !
> I'm removing this stuff as its just an attempt to promote the Bates method, not an attempt at 'neutrality' It is an attempt to give valueble information about amblyopia.. I am quite sure a lot of people will find this information very interesting not an attempt at 'neutrality' Again that is your opinion Objective people will say you are not neutral by removing this information.
> When they use the term 'lazy eye', ophthalmologists don't mean the eye is actually 'lazy'. Or, indeed, that it is 'tense' and equally meaningless comment. don't mean the eye is actually 'lazy'. I do understand why ophthalmologists call amblyopia lazy eye. It looks like a ambliopic eye is lazy from the ophthalmologists point of view. But when you really understand and know the cause, a far better name is tense eye. Now ophthalmologists give on a subjecitve level false information that the eye is not working hard enough. And make it very hard for the public to find the real reason and solutions. In other words This is very valueable information with a reference everybody can check, When you think the ophthalmologists are right and the natural vision improvement movement is wrong about the cause (?) you are not neutral. Do ophthalmologists say something about the cause of amblyopia by the way ? Or are they vague ? Seeyou 22:06, 29 January 2007 (UTC)
1. Again, why is the reference vague ? Please explain.with arguments ?
2. Fact : Objective people will say you are not neutral by removing this information. True or untrue ?
The name lazy eye makes it very hard for the public to find the real reason and solutions. In other words This is very valueable information with a reference to a book everybody can check when they are interested, When you think the ophthalmologists are right and the natural vision improvement movement is wrong about the cause (?) you are not neutral. We should be very happy wikipedia can allow this information to reach the public.
Q : 1) Because you haven't defined what 'lazy', 'poor' and 'tense' mean and what they refer to. I have not defined anything ! A : I have given a citation from a book with a opposite point of view. ( A lazy eye needs to work harder, a tense eye needs relaxation. )
This information is "valuable" only to people selling useless products supposed to help that only have anecdotal evidence to support their use, not valuable to people with amblyopia. A : That is your opinion. There also people selling glasses and making a lots of money. This is no argument.
"Tense" is not a better word that "lazy", because neither of them are helpful or descriptive. A : Tense is far better than lazt because is gives information about the cause and the solution. Lazy describes only the symptom
Mainstream ophthalmology and neuroscience (my own particulart interest in this topic) haven't found a cure (yet!), but I believe they one day will. A : I don’t think they will find a cure. They have got the wrong starting point, but you never know they might read this.!
The "natural vision movement" hasn't found a cure either but gives false hope to people by selling its books and tapes on purely anecdotal support rather than proper science. A : That is your opinion. My opinion is the opposite.
I don’t think we will ever come to an agreement. Let’s ask others what they think about the referenced information lazy eye or tense eye :
<ref>
tag has too many names (see the
help page)..The section in question [1] is original research and should be kept out of the article. There is also considerations about how much weight is placed on Bate's methods; it appears to me that a wikilink is enough, and a section of a paragraph would give it undue weight. It doesn't matter if Bate's method is true or not, Wikipedia is not the place to debate what is true and what is false, but to report the facts (as WP:Verifiability defines them) and POVs as POVs to the extent of the significance of those who take them (of course, you can add as much stuff about Bate's method in the article of that name, but not on this page). I suggest anyone wanting to add the section back to revise Wikipedia policies first. RB 9 7 2 23:42, 3 February 2007 (UTC)
third party, Thank you. Now I understand. From the point of view of the public the source is not reliable. Still I hope the public will rebel and check the source with an open mind. Seeyou. Seeyou 21:25, 4 February 2007 (UTC)
Amblyopia is not "lazy eye". Strabismus is. I have strabismic amblyopia. That is, as a child my strabismus (now cured) led to amblyopia (not cured). I have not had "lazy eye" since my childhood even though I still suffer from amblyopia. I thus suggest that the appositive , or "lazy eye", be removed from the article's first sentence. -- 198.203.175.175 14:55, 11 April 2007 (UTC)
It might be an idea to remove the "lazy eye" quote but in the 50's, that is what is was called. I was diagnosed too late for the patch to work because I was "too aware of my looks" and kept taking the patch off till everyone gave up. I still remember to this day when the Doctor told me that I had "Lazy Eye." I would hate for this page to be missed by those searching for "Lazy Eye" even if that is not the correct definition. Larewill50 ( talk) 02:42, 26 January 2010 (UTC)
Hi there,
Just read the article on amblyopia. It raises as many questions as it answers, mainly because so many difficult terms are used but not explained, at least no by wikipedia. These terms seem to be linked to non existing articles :(
Sitz
I don't know where to put this comment, but I have amblyopia and I have depth perception AND I am able to see autostereogams with no difficulty. I was diagnosed at age 7 and I had my first exposure to an autostereogram at age 8. It took a little while for me to understand how to see them, but after a friend helped me, I have been able to see them with no problem. I'm almost 24 now. I wonder how many amblyopes can actually see autostereograms and stereograms. Perhaps mine is not that servere of a case (although I do have 4 diopters [sp?] of astigmatisim in my amblyopic eye). Jcsavestheday ( talk) 05:36, 19 February 2009 (UTC)jcsavestheday
- this is a clear demonstration that amblyopia and strabismus are two totally different conditions. Related yes, but quite different. ( Doc Adam ( talk) 00:42, 18 April 2013 (UTC))
People who have never had binocular vision don't understand what it would be like to have it -- and vice versa. This article seems to be full of the normal view of people who do not have amblyopia, who are sure it must be a terrible curse to have it. But, actually, people with amblyopia can have depth perception. It would be interesting to find an accurate description of what difference binocular vision actually makes -- but it would probably have to be written by researchers who don't have binocular vision... - 69.87.203.221 23:14, 11 September 2007 (UTC)
Absolutely!!!!
I'm Amblyopic too, and have no problem with depth perception. It's actually because the brain learns to interpolate from 2d to 3d and doesn't need the other eye. When I did geology at college I had to interpolate 2d maps in 3d and was absolutely the 'ace' of the class - but I couldn't explain what I could 'see' in my head. — Preceding unsigned comment added by 95.146.117.230 ( talk) 23:33, 24 April 2012 (UTC)
I have to disagree. I have amblyopia, discovered when I was around 6, too late for correction. (I'm 52 now.) I occasionally have trouble with depth perception, pouring water outside of a glass, for example. I saw a neuro-opthalmologist some years age because I have another neurological problem, and he explained that while I don't have actual depth perception, there is an adaptation that, in most cases, gets around it. The brain does not "interpolate," because the brain doesn't know what 3D looks like. Kirkmc ( talk) 07:58, 25 April 2012 (UTC)
Well as long as we're being anecdotal, I have strabismus, and therefore no depth perception. But every once in a while, under ideal conditions, the 3D effect kicks in. So the perceptual brain circuits must still be there, just the visual processing failed to develop. I would think there's room there for more research - and conceivably another Nobel! ( Doc Adam ( talk) 00:36, 18 April 2013 (UTC))
Is there a reason why Lazy eye isn't redirected here? It would appear that this should be so from reading the other article. · AndonicO Talk 22:02, 15 November 2007 (UTC)
Let me explain why I wholesale removed the section on prozac as a potential "cure"...
This is one study. And its in mice not humans. And it involves a drug. And this drug has recently been shown to not really have any great antidepressant effects. That means that producers of prozac could potentially be losing their "depressed kids" market and may be scouting about for some other group to sell it to.
Finally, and most importantly, I would feel pretty bad if, based on this flimsy evidence, amblyopic kids started taking Mum's prozac because they didn't like wearing a patch for two hours a day. I suggest we let scientists do their job and test this hypothesis before proclaiming it as a new "cure". The jury is very much still out on this one. One swallow does not a summer make... Famousdog ( talk) 18:28, 1 May 2008 (UTC)
This is one study. And its in mice not humans. And it involves a drug. And this drug has recently been shown to not really have any great antidepressant effects. That means that producers of prozac could potentially be losing their "depressed kids" market and may be scouting about for some other group to sell it to.
Obviously you have some baggage regarding fluoxetine. The drug is favorable for as a first line anti-depressant the problem is patients poor combliance to the drug since it takes 4-6 weeks for onset of effects. —Preceding unsigned comment added by 72.138.6.203 ( talk) 00:59, 8 April 2009 (UTC)
I looked for help or a cure but there is none. Lasik surgery wants nothing to do with me. 3 eye doctors tell me oh im fine. I'm here as an Amblyopic representitive to ask where then is the knowledge to repair this problem ! I am now 33 and have 20% vision in my right eye. I am especially angry that this disease is called " LAZY EYE" People say "well i never knew your eye was crooked" and I have to educate them on a subject that has no information,charts,graphs,websites,links,photosw,groups or any info at all. .....ect, No one seems to know or care about this and it affects my life every day in all that I do. I don't know about you all but I need to see! Please help research this disease and find a cure for it and recognition as an extremely depressing disease and immensely underated, understudied seriously oppressing handicap. aka blindness. If anyone knows any info that would be great. I should be eligible for ssd? Anyone know? Thank You... Cosmos0001 ( talk)Blindness I can be part of any study anyone is trying on this disease. I want to see!
04:03, 2 June 2009 (UTC)
Looking at the treatment and prognosis section where it talks about the Enfant system, the tone does not seem to me to be encyclopedic - the section only discusses the drawbacks of the conventional detection method, and the pros of the Enfant system, using terms like "breakthrough technology" and a list of features of the system which does not seem to be appropriate for an article about one of the conditions that the system detects. While it may merit a mention on the page as a detection method, with some reasoning provided, I think this might warrant a rewrite (by someone more knowledgeable on the topic than myself, probably). Interestingly average ( talk) 18:37, 21 August 2009 (UTC)
The lazy eye is an effect from the frontal lobe, correct? What about the limbic system? The frontal lobe is the first stage of birth. All psychology, Autism/Aspergers comes from the frontal lobe, if limbic system. It is known that epilepsy, diabetes (psychologically if genetic) can assist/partner the lazy eye? Thank you for your time, God bless you —Preceding unsigned comment added by Musketeer41 ( talk • contribs) 17:23, 14 July 2010 (UTC)
I just removed 2 references from the page:
Destructoid: Mario Kart Saved Child's Eyesight [3]
Neither meets the requirements of WP:RS, as gamepolitics is a blog and the other boasts of having "unfiltered and unedited" commentary. Both seem to be referring to legitimate medical studies, which would be reliable. So if we can find those, then we can go ahead and keep the info. I left the info in the page for now with cite needed tags, as it seems likely that the info can be added. I can't search PubMed or the equivalent, but if I have time and inclination later I'll try Google Scholar. Anyone else, of course, is welcome to do the same. Qwyrxian ( talk) 11:45, 27 September 2010 (UTC)
This could be a useful ref.
Doc James ( talk · contribs · email) (if I write on your page reply on mine) 14:01, 12 April 2014 (UTC)
I added a short paragraph concerning amblyopia and neuroplasticity to update the public on current molecular biology research that is of interest to amblyopia. All sources are recently published and are from reputable, peer-reviewed scientific journals (please see citations). Links are included if a reader would like to know more about the proteins currently being researched. — Preceding unsigned comment added by Jmb8539 ( talk • contribs) 22:14, 10 December 2014 (UTC)
It is written that: "In amblyopia visual stimulation either fails to be or is poorly transmitted through the optic nerve to the brain for a continuous period of time" This can be understood as if the cause of amblyopia was in a problem of optic nerve. This is obviously false. There is no research backing up this claim, while there are thousands of papers showing this has a cortical cause (and the debate is rather about the pure implication of V1 or of other areas, like extra-striate visual areas or LGN: see Levi (2013) for a review). This is important to correct that, given I have seen the error spreading in non-academic websites or translation of the wikipedia webpage in other languages. This is going to far and it is time to stop the bleeding. I think the original intent of the sentence is to explain that whenever the brain does not receive visual stimulation for a continuous period of time, there is a risk for amblyopia. I am open to discussion, but there is a consensus in academia on that so there should be none. -- Adrien16 ( talk) 15:46, 20 July 2015 (UTC)
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Evidence regarding treatments for adults is poor.
This could have a direct meaning: we have scant evidence about efficacy in adults.
Or it could have a loosey-goosey meaning: Evidence attests that efficacy in adults is poor. (Careless speakers in oral context spin out this kind of sentence all the time.)
Whichever it is, this phrasing strikes me as is too oblique. — MaxEnt 17:21, 27 February 2019 (UTC)
With respect to speciality, would one send someone with amblyopia to a neurology or an ophthalmologist? Doc James ( talk · contribs · email) 17:28, 18 February 2020 (UTC)
I suggest adding a "See Also" section with links to related conditions such as diplopia, myopia and hyperopia. What do you think? — Preceding unsigned comment added by 131.150.125.128 ( talk) 19:09, 8 June 2021 (UTC)
Ideal sources for Wikipedia's health content are defined in the guideline
Wikipedia:Identifying reliable sources (medicine) and are typically
review articles. Here are links to possibly useful sources of information about Amblyopia.
|
This article is rated B-class on Wikipedia's
content assessment scale. It is of interest to the following WikiProjects: | |||||||||||||||||||||||||||||||
|
It seems like a tough question whenever one wants to answer precisely (is it more 1% or more 5%) because it depends on the country, the age, and mostly the criterion chosen (one line, two line difference? including all amblyogenic causes or not?). However, when one is claiming that it is between 1 and 5%, ALL studies agree (in western countries). So I don't think so we can go wrong on this one. They agree no matter what is the age or the criterion, but the range can go to 10% when including non-western countries.
Secondary sources:
-- Adrien Chopin ( talk) 07:52, 21 April 2016 (UTC)
It was quite ironic that Amblyopia is one of the very few diseases that is not tamed enough by the human intelligence. It almost goes unnoticed at the very age of the victim, when it can be best controlled. It is good that children are lucky enough to have wonderful treatment regimens available to them these days, if only, when detected in time, that is in their very early years, but the stigma and pain that is associated with Adult Amblyopia is particularly unbearable. I came to know that I am one of the very few people who fell to this seemingly innocuous disease(rather poor vision condition - doctors won't give up to call it a disease) when I was first disqualified in 'Army Selections'. That was a blow to my career as I always dreamt of becoming an 'Officer in Uniform' right from my childhood. Then started my ordeal, it forced me to change my goals, take up unplanned ventures, constantly irritated me now and then, and more than anything else, I am very much depressed to imagine the fact that one day I will become a one-eyed jack.
While it is extremely painful to learn that there is no proven treatment for the adults, the plight of the people like me in the still developing countries is un-imaginable, where nothing is being done effectively, even the diagnosis of this bloody disease( let me vent my anger).
My Dear Brothers Of The World Who Are In The Same Boat With Me: We share the slogan with the victims of the most dreaded disease(or ???condition) on this earth AIDS:
DOCTORS OF THE WORLD: DO SOMETHING AND SAVE US before it is too late.
Does Amblyopia have anything to do with Ocular Dominance?
Ocular Dominance Wikipedia link:
http://en.wikipedia.org/wiki/Ocular_dominance
Amblyopia has a lot to do with ocular dominance, though not necessarily in a clear-cut way. Monocular occlusion during development is a well-known way to cause amblyopia, and causes the ocular dominance columns of cortex for the dominant eye of the amblyope to dominate that of the amblyopic eye. Behaviorally, amblyopia - which has a poor medical definition - exhibits symptoms that suggest information loss in transmission from eye to cortex because of poor development. That information loss could be caused by the mechanics of the screwed up distribution of ocular dominance cells, which extends in a loop from the eye through the lateral geniculate nucleus, and into the cortex, as well as into the pons which controls certain automatic tracking activities.<spetz>. 216.175.188.98 22:17, 28 February 2007 (UTC)
The controversial aspect is covered by the bates method article. It now looks as if the claim of some is controversial. Seeyou 22:03, 18 January 2007 (UTC)
About the deletion lazy eye or tense eye. WIkipedia also has the policy of a neutral point of view. That means when there are substantial bodies of opposing opinions, they all need to be presented. The formula is "A says B about C." You can't say in the article "mainstream medicine is bunk." You can't say in a vague sort of way "Bates supporters think mainstream medicine is bunk." You can say "so-and-so, in his book Thus-and-such, says on page 234, 'mainstream medicine is bunk.'". Seeyou 18:50, 25 January 2007 (UTC)
> Look your vague references to eyes being 'tense' as opposed to 'lazy' or 'poor' suggest that you don't know what amblyopia is. Why is the reference vague, please explain. suggest that you don't know what amblyopia is ? That is your opinion !
> I'm removing this stuff as its just an attempt to promote the Bates method, not an attempt at 'neutrality' It is an attempt to give valueble information about amblyopia.. I am quite sure a lot of people will find this information very interesting not an attempt at 'neutrality' Again that is your opinion Objective people will say you are not neutral by removing this information.
> When they use the term 'lazy eye', ophthalmologists don't mean the eye is actually 'lazy'. Or, indeed, that it is 'tense' and equally meaningless comment. don't mean the eye is actually 'lazy'. I do understand why ophthalmologists call amblyopia lazy eye. It looks like a ambliopic eye is lazy from the ophthalmologists point of view. But when you really understand and know the cause, a far better name is tense eye. Now ophthalmologists give on a subjecitve level false information that the eye is not working hard enough. And make it very hard for the public to find the real reason and solutions. In other words This is very valueable information with a reference everybody can check, When you think the ophthalmologists are right and the natural vision improvement movement is wrong about the cause (?) you are not neutral. Do ophthalmologists say something about the cause of amblyopia by the way ? Or are they vague ? Seeyou 22:06, 29 January 2007 (UTC)
1. Again, why is the reference vague ? Please explain.with arguments ?
2. Fact : Objective people will say you are not neutral by removing this information. True or untrue ?
The name lazy eye makes it very hard for the public to find the real reason and solutions. In other words This is very valueable information with a reference to a book everybody can check when they are interested, When you think the ophthalmologists are right and the natural vision improvement movement is wrong about the cause (?) you are not neutral. We should be very happy wikipedia can allow this information to reach the public.
Q : 1) Because you haven't defined what 'lazy', 'poor' and 'tense' mean and what they refer to. I have not defined anything ! A : I have given a citation from a book with a opposite point of view. ( A lazy eye needs to work harder, a tense eye needs relaxation. )
This information is "valuable" only to people selling useless products supposed to help that only have anecdotal evidence to support their use, not valuable to people with amblyopia. A : That is your opinion. There also people selling glasses and making a lots of money. This is no argument.
"Tense" is not a better word that "lazy", because neither of them are helpful or descriptive. A : Tense is far better than lazt because is gives information about the cause and the solution. Lazy describes only the symptom
Mainstream ophthalmology and neuroscience (my own particulart interest in this topic) haven't found a cure (yet!), but I believe they one day will. A : I don’t think they will find a cure. They have got the wrong starting point, but you never know they might read this.!
The "natural vision movement" hasn't found a cure either but gives false hope to people by selling its books and tapes on purely anecdotal support rather than proper science. A : That is your opinion. My opinion is the opposite.
I don’t think we will ever come to an agreement. Let’s ask others what they think about the referenced information lazy eye or tense eye :
<ref>
tag has too many names (see the
help page)..The section in question [1] is original research and should be kept out of the article. There is also considerations about how much weight is placed on Bate's methods; it appears to me that a wikilink is enough, and a section of a paragraph would give it undue weight. It doesn't matter if Bate's method is true or not, Wikipedia is not the place to debate what is true and what is false, but to report the facts (as WP:Verifiability defines them) and POVs as POVs to the extent of the significance of those who take them (of course, you can add as much stuff about Bate's method in the article of that name, but not on this page). I suggest anyone wanting to add the section back to revise Wikipedia policies first. RB 9 7 2 23:42, 3 February 2007 (UTC)
third party, Thank you. Now I understand. From the point of view of the public the source is not reliable. Still I hope the public will rebel and check the source with an open mind. Seeyou. Seeyou 21:25, 4 February 2007 (UTC)
Amblyopia is not "lazy eye". Strabismus is. I have strabismic amblyopia. That is, as a child my strabismus (now cured) led to amblyopia (not cured). I have not had "lazy eye" since my childhood even though I still suffer from amblyopia. I thus suggest that the appositive , or "lazy eye", be removed from the article's first sentence. -- 198.203.175.175 14:55, 11 April 2007 (UTC)
It might be an idea to remove the "lazy eye" quote but in the 50's, that is what is was called. I was diagnosed too late for the patch to work because I was "too aware of my looks" and kept taking the patch off till everyone gave up. I still remember to this day when the Doctor told me that I had "Lazy Eye." I would hate for this page to be missed by those searching for "Lazy Eye" even if that is not the correct definition. Larewill50 ( talk) 02:42, 26 January 2010 (UTC)
Hi there,
Just read the article on amblyopia. It raises as many questions as it answers, mainly because so many difficult terms are used but not explained, at least no by wikipedia. These terms seem to be linked to non existing articles :(
Sitz
I don't know where to put this comment, but I have amblyopia and I have depth perception AND I am able to see autostereogams with no difficulty. I was diagnosed at age 7 and I had my first exposure to an autostereogram at age 8. It took a little while for me to understand how to see them, but after a friend helped me, I have been able to see them with no problem. I'm almost 24 now. I wonder how many amblyopes can actually see autostereograms and stereograms. Perhaps mine is not that servere of a case (although I do have 4 diopters [sp?] of astigmatisim in my amblyopic eye). Jcsavestheday ( talk) 05:36, 19 February 2009 (UTC)jcsavestheday
- this is a clear demonstration that amblyopia and strabismus are two totally different conditions. Related yes, but quite different. ( Doc Adam ( talk) 00:42, 18 April 2013 (UTC))
People who have never had binocular vision don't understand what it would be like to have it -- and vice versa. This article seems to be full of the normal view of people who do not have amblyopia, who are sure it must be a terrible curse to have it. But, actually, people with amblyopia can have depth perception. It would be interesting to find an accurate description of what difference binocular vision actually makes -- but it would probably have to be written by researchers who don't have binocular vision... - 69.87.203.221 23:14, 11 September 2007 (UTC)
Absolutely!!!!
I'm Amblyopic too, and have no problem with depth perception. It's actually because the brain learns to interpolate from 2d to 3d and doesn't need the other eye. When I did geology at college I had to interpolate 2d maps in 3d and was absolutely the 'ace' of the class - but I couldn't explain what I could 'see' in my head. — Preceding unsigned comment added by 95.146.117.230 ( talk) 23:33, 24 April 2012 (UTC)
I have to disagree. I have amblyopia, discovered when I was around 6, too late for correction. (I'm 52 now.) I occasionally have trouble with depth perception, pouring water outside of a glass, for example. I saw a neuro-opthalmologist some years age because I have another neurological problem, and he explained that while I don't have actual depth perception, there is an adaptation that, in most cases, gets around it. The brain does not "interpolate," because the brain doesn't know what 3D looks like. Kirkmc ( talk) 07:58, 25 April 2012 (UTC)
Well as long as we're being anecdotal, I have strabismus, and therefore no depth perception. But every once in a while, under ideal conditions, the 3D effect kicks in. So the perceptual brain circuits must still be there, just the visual processing failed to develop. I would think there's room there for more research - and conceivably another Nobel! ( Doc Adam ( talk) 00:36, 18 April 2013 (UTC))
Is there a reason why Lazy eye isn't redirected here? It would appear that this should be so from reading the other article. · AndonicO Talk 22:02, 15 November 2007 (UTC)
Let me explain why I wholesale removed the section on prozac as a potential "cure"...
This is one study. And its in mice not humans. And it involves a drug. And this drug has recently been shown to not really have any great antidepressant effects. That means that producers of prozac could potentially be losing their "depressed kids" market and may be scouting about for some other group to sell it to.
Finally, and most importantly, I would feel pretty bad if, based on this flimsy evidence, amblyopic kids started taking Mum's prozac because they didn't like wearing a patch for two hours a day. I suggest we let scientists do their job and test this hypothesis before proclaiming it as a new "cure". The jury is very much still out on this one. One swallow does not a summer make... Famousdog ( talk) 18:28, 1 May 2008 (UTC)
This is one study. And its in mice not humans. And it involves a drug. And this drug has recently been shown to not really have any great antidepressant effects. That means that producers of prozac could potentially be losing their "depressed kids" market and may be scouting about for some other group to sell it to.
Obviously you have some baggage regarding fluoxetine. The drug is favorable for as a first line anti-depressant the problem is patients poor combliance to the drug since it takes 4-6 weeks for onset of effects. —Preceding unsigned comment added by 72.138.6.203 ( talk) 00:59, 8 April 2009 (UTC)
I looked for help or a cure but there is none. Lasik surgery wants nothing to do with me. 3 eye doctors tell me oh im fine. I'm here as an Amblyopic representitive to ask where then is the knowledge to repair this problem ! I am now 33 and have 20% vision in my right eye. I am especially angry that this disease is called " LAZY EYE" People say "well i never knew your eye was crooked" and I have to educate them on a subject that has no information,charts,graphs,websites,links,photosw,groups or any info at all. .....ect, No one seems to know or care about this and it affects my life every day in all that I do. I don't know about you all but I need to see! Please help research this disease and find a cure for it and recognition as an extremely depressing disease and immensely underated, understudied seriously oppressing handicap. aka blindness. If anyone knows any info that would be great. I should be eligible for ssd? Anyone know? Thank You... Cosmos0001 ( talk)Blindness I can be part of any study anyone is trying on this disease. I want to see!
04:03, 2 June 2009 (UTC)
Looking at the treatment and prognosis section where it talks about the Enfant system, the tone does not seem to me to be encyclopedic - the section only discusses the drawbacks of the conventional detection method, and the pros of the Enfant system, using terms like "breakthrough technology" and a list of features of the system which does not seem to be appropriate for an article about one of the conditions that the system detects. While it may merit a mention on the page as a detection method, with some reasoning provided, I think this might warrant a rewrite (by someone more knowledgeable on the topic than myself, probably). Interestingly average ( talk) 18:37, 21 August 2009 (UTC)
The lazy eye is an effect from the frontal lobe, correct? What about the limbic system? The frontal lobe is the first stage of birth. All psychology, Autism/Aspergers comes from the frontal lobe, if limbic system. It is known that epilepsy, diabetes (psychologically if genetic) can assist/partner the lazy eye? Thank you for your time, God bless you —Preceding unsigned comment added by Musketeer41 ( talk • contribs) 17:23, 14 July 2010 (UTC)
I just removed 2 references from the page:
Destructoid: Mario Kart Saved Child's Eyesight [3]
Neither meets the requirements of WP:RS, as gamepolitics is a blog and the other boasts of having "unfiltered and unedited" commentary. Both seem to be referring to legitimate medical studies, which would be reliable. So if we can find those, then we can go ahead and keep the info. I left the info in the page for now with cite needed tags, as it seems likely that the info can be added. I can't search PubMed or the equivalent, but if I have time and inclination later I'll try Google Scholar. Anyone else, of course, is welcome to do the same. Qwyrxian ( talk) 11:45, 27 September 2010 (UTC)
This could be a useful ref.
Doc James ( talk · contribs · email) (if I write on your page reply on mine) 14:01, 12 April 2014 (UTC)
I added a short paragraph concerning amblyopia and neuroplasticity to update the public on current molecular biology research that is of interest to amblyopia. All sources are recently published and are from reputable, peer-reviewed scientific journals (please see citations). Links are included if a reader would like to know more about the proteins currently being researched. — Preceding unsigned comment added by Jmb8539 ( talk • contribs) 22:14, 10 December 2014 (UTC)
It is written that: "In amblyopia visual stimulation either fails to be or is poorly transmitted through the optic nerve to the brain for a continuous period of time" This can be understood as if the cause of amblyopia was in a problem of optic nerve. This is obviously false. There is no research backing up this claim, while there are thousands of papers showing this has a cortical cause (and the debate is rather about the pure implication of V1 or of other areas, like extra-striate visual areas or LGN: see Levi (2013) for a review). This is important to correct that, given I have seen the error spreading in non-academic websites or translation of the wikipedia webpage in other languages. This is going to far and it is time to stop the bleeding. I think the original intent of the sentence is to explain that whenever the brain does not receive visual stimulation for a continuous period of time, there is a risk for amblyopia. I am open to discussion, but there is a consensus in academia on that so there should be none. -- Adrien16 ( talk) 15:46, 20 July 2015 (UTC)
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Evidence regarding treatments for adults is poor.
This could have a direct meaning: we have scant evidence about efficacy in adults.
Or it could have a loosey-goosey meaning: Evidence attests that efficacy in adults is poor. (Careless speakers in oral context spin out this kind of sentence all the time.)
Whichever it is, this phrasing strikes me as is too oblique. — MaxEnt 17:21, 27 February 2019 (UTC)
With respect to speciality, would one send someone with amblyopia to a neurology or an ophthalmologist? Doc James ( talk · contribs · email) 17:28, 18 February 2020 (UTC)
I suggest adding a "See Also" section with links to related conditions such as diplopia, myopia and hyperopia. What do you think? — Preceding unsigned comment added by 131.150.125.128 ( talk) 19:09, 8 June 2021 (UTC)