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please do so. If it no longer meets these criteria, you can
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There is a problem with the lower EKG image and its legend. The legend refers to two panels, one after treatment. The figure shows three panels for three different classes of the syndrome.
98.64.68.45 ( talk) 12:24, 8 October 2009 (UTC)
I agree with the poster below that the subjects should not be merged because their definitions differ. In addition, the disorders described as SUD (or SUNDS, Sudden Unexplained Nocturnal Death Syndrome) such as Bangungot can be classified as ethnomedical disorders. The description of the phenomena and traditional cures will undoubtedly differ in each case. To lump any or all of them together as Brugada seems reductionist and ethnocentric. Doing so would probably not only be scientifically wrong, but gives precedence to a biological cause and Western medical remedy (as found in the Brugada article) rather than allowing for a focus on the psycho-social reality of each disorder and valuable discussions of traditional remedies. I would suggest that there should be a section on linked ethnomedical disorders in the main Brugada article and that there should be links to each of the disorders' articles at the end of the Brugada article.
This link should take you to a 2002 article in Human Molecular Genetics which indicates a genetic link between SUNDS and Brugada: hmg.oxfordjournals.org/cgi/reprint/11/3/337.pdf
On a related note, More information on some of the ethnomedical disorders linked to SUNDS (particularly in Hmong men) can be found below. SUNDS has long been linked to sleep paralysis. See:
1) Hufford, David J. The Terror That Comes in the Night. Philadelphia: University of Pennsylvania Press, 1982. 2) Adler, Shelly R. "The Role of the Nightmare in Hmong Sudden Unexpected Nocturnal Death Syndrome: A Folkloristic Study of Belief and Health." Ph.D. diss., University of California--Los Angeles, 1991, 48-61. 3) Adler, Shelly R. "Sudden Unexpected Nocturnal Death Syndrome among Hmong Immigrants: Examining the Role of the 'Nightmare'," Journal of American Folklore 104 (1991): 54-71 Lorist 21:34, 6 August 2007 (UTC)
..one may call that an amateur page...
... and just what have _you_ written?
Brugada syndrome is a well defined entity, whereas the other ones are more ill-defined. There is no doubt that Brugada syndrome is a part of the others but other diseases is also part of SUDS and sudden death in asia. Therefore the subjects should not be merged.
A good reference:
1. Priori SG, Aliot E, Blomstrom-Lundqvist C, Bossaert L, Breithardt G, Brugada P, Camm AJ, Cappato R, Cobbe SM, Di Mario C, Maron BJ, McKenna WJ, Pedersen AK, Ravens U, Schwartz PJ, Trusz-Gluza M, Vardas P, Wellens HJ, Zipes DP. Task Force on Sudden Cardiac Death of the European Society of Cardiology. Eur Heart J. 2001 Aug;22(16):1374-450. ( Medline abstract)
Ksheka 11:54, Aug 24, 2004 (UTC)
Hi, I am an ICD patient myself as a result of going into ventricular fibrillation in my sleep in 2004. This is also known as SADS (Sudden arrhythmic death syndrome).
I have been genetically tested for the genes related to Brugada in 2005. The result was negative but I am having more genetic tests done. I have just recently had a blood sample taken for the second time. Hopefully they can research more into this. I have more information regarding brugada syndrome and my doctor has already met Dr. Brugada himself.
I would like to add more information about testing for Brugada. There is a drug called "ajmaline" which I was tested with to see if any abnormalities in my ECG showed up. Perhaps I could add this to the article and other information too? Smsmasters 09:02, 28 June 2007 (UTC)
All Class Ic "sodium current blocking agents" could be used to reveal hidden ECG abnormalities to diagnose this entity. However, prognostic significance of this test is quite questionable, indeed people without any VF episodes or spontaneous ECG changes has very little chance to develop a sudden cardiac death. Also, a new "old" drug is currently popular in treatment of Brugada syndrome: Quinidine. So I added some info about this topic with reference. —Preceding unsigned comment added by Lord Xar ( talk • contribs) 22:15, 18 September 2007 (UTC)
You could add a link to [www.brugadadrugs.org] on the wiki, a site discussing drugs associated with Brugada syndrome and lists of drugs to avoid.
Petertje puk ( talk) 19:16, 29 June 2009 (UTC)
Hello fellow Wikipedians,
I have just modified 3 external links on Brugada syndrome. Please take a moment to review my edit. If you have any questions, or need the bot to ignore the links, or the page altogether, please visit this simple FaQ for additional information. I made the following changes:
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Hi @ Doc James:, I have just realised that the edits I made to the Brugada page yesterday inadvertently deleted the infobox, sorry.
I see that you in addition to restoring the infobox, you reverted the changes I made to the lead, with your comment being MEDMOS. I'd be grateful if you could help me understand in what way my version of the lead (pasted below) falls foul of MEDMOS - I felt it was an improvement as it avoided technical terms like 'vagal tone' which may be challenging for a lay reader to understand, while summarising the key points in the main body of the article below. I appreciate that you have a lot of Wiki experience and welcome your feedback. Thanks PeaBrainC ( talk) 21:49, 17 January 2018 (UTC)
@ Doc James:, PeaBrainC ( talk) 20:07, 18 January 2018 (UTC)
Brugada syndrome (BrS) is a
genetic condition which results in abnormal electrical activity within the
heart, increasing the risk of
abnormal heart rhythms and
sudden cardiac death.
[1] These abnormal heart rhythms often occur at rest or after a heavy meal, and can be triggered by a
fever, excessive alcohol, or certain medications.
[1] The symptoms of Brugada syndrome include
fainting but those affected may not have any symptoms at all.
[2]
As an inherited condition, about a quarter of those affected by Brugada syndrome will have a relative who also has the condition. [1] It may be caused by mutations in the SCN5A gene responsible for the cardiac sodium channel, although mutations causing the condition have also been found in other genes.
Brugada syndrome is typically diagnosed using an electrocardiogram (ECG), although medications such as ajmaline may be required to produce the characteristic ECG pattern. [1] There is no cure for Brugada syndrome, but the condition may be treated using an implantable cardioverter defibrillator (ICD). [3] Medications to help control the abnormal heart rhythms include isoproterenol in those who are acutely unstable, and quinidine. [4] The family members of a patient with Brugada syndrome may require testing for the condition. [4]
Were does this ref mention after meals or during sleep?
Not seeing "while
quinidine may be used in the longer term" in
https://rarediseases.org/rare-diseases/brugada-syndrome/ Found the ref and added it to the sentence in question.
Doc James ( talk · contribs · email) 18:49, 9 March 2018 (UTC)
GA toolbox |
---|
Reviewing |
Reviewer: Natureium ( talk · contribs) 16:25, 11 July 2018 (UTC)
I've never done a GAN review before, so please let me know (politely) if I'm doing anything wrong.
GA review – see WP:WIAGA for criteria
Mechanism: heart racing -> rapid heart rate : Done
PeaBrainC (
talk)
17:19, 13 July 2018 (UTC)
Hi @ Natureium:, thanks for reviewing this article. It's only my 2nd GAN but to my inexperienced eyes looks like you've done a good job. I will get on with making the amendments you have suggested and mark them as done on this page as I go. It may take a few days - busy times in the real world! PeaBrainC ( talk) 09:48, 12 July 2018 (UTC)
Sentence 4 begins: The abnormal heart rhythm often occurs. The The implies that the reader is to know which abnormal hear rhythm we are talkin about. But no abnormal heart rhythm has been mentioned, I think. That is, I think that there are heart disorders that are not abnomal heart rhythms. Even if there aren't, we should not assume that all readers know this.--
Ettrig (
talk) 20:57, 12 July 2018 (UTC) Done
PeaBrainC (
talk)
17:06, 13 July 2018 (UTC)
Hi Yanping, I wonder if we could have a discussion on the talkpage about our differences of opinion regarding the best form of language to use in the article. I don't want to edit war and am loathe to change your edits again without consensus, so I'm bringing it to the table here.
In your recent edit summaries you have suggested that "this page should be written for academics and professionals, if laypeople don’t know what sodium channels are they’re supposed to follow wikilinks" and that "this is a science article, not a brochure for patients". All the manuals of style suggest that articles should be written in a style suitable for a non specialist. From WP:BATTLE - "Texts should be written for everyday readers, not just for academics", and "While wikilinks should be provided for advanced terms and concepts in that field, articles should be written on the assumption that the reader will not or cannot follow these links, instead attempting to infer their meaning from the text."
In addition to our differences of opinion on writing style there are questions to be raised regarding the accuracy of what you have changed. Whether the prose is more or less technically oriented, it needs to be correct. For example, SCN5A is a gene. NaV1.5 is the protein forming the alpha subunit of the cardiac sodium channel that SCN5A encodes. INa is the current that NaV1.5 carries. These terms should not be mixed up as was done in your recent set of edits. Furthermore, Brugada has nothing to do with the late sodium current - increased late INa is responsible for the LQT3 form of long QT syndrome. Brugada is an oligogenic disorder but the association with sodium is entirely to do with a reduction in the peak current. It is wrong to say that BrS is due to mutations in SCN5A - a proportion of cases of BrS are associated with SCN5A mutations but in the majority no causative gene can be found.
Comparing your most recent version of the first paragraph of the causes section:
"The individual cells of the heart communicate with each other with electrical signals, and these electrical signals are disrupted in those with Brugada syndrome due to mutations in SCN5A, a cardiac sodium channel which is responsible for depolarizing the cardiomyocyte during phase 0 of the cardiac action potential by permitting the early component of the sodium current as well as permitting a residual current lasting throughout the low-conductance phases of repolarization, known as the late sodium current. Many of the genetic mutations that have subsequently been described in association with Brugada syndrome influence the early and late components of the sodium current in some way, often differentially, or affect other ionic currents."
to the previous version:
"The individual cells of the heart communicate with each other with electrical signals, and these electrical signals are disrupted in those with Brugada syndrome. As a genetic condition, the syndrome is ultimately caused by changes to a person's DNA, known as genetic mutations. The first mutations described in association with Brugada syndrome were in a gene responsible for a protein or ion channel that controls the flow of sodium ions through the cell membrane of heart muscle cells – the cardiac sodium channel. Many of the genetic mutations that have subsequently been described in association with Brugada syndrome influence the sodium current in some way, or affect other ionic currents."
I feel that the earlier version was more accurate and worded in a way more appropriate to a non-specialist audience. Like I say, I'm not going to revert again, but would appreciate the opinions of others who have previously shown an interest in the article. @ Yanping Nora Soong: @ Doc James: @ TylerDurden8823:. Thanks, PeaBrainC ( talk) 17:00, 14 February 2019 (UTC)
"As early as December 1981, the Center for Disease Control described sudden cardiac death during sleep of predominantly male Southeast Asian refugees. [5] According to the CDC report, "The abruptness of the deaths reported here is compatible with a cardiac dysrhythmia ... but the underlying mechanism remains unclear." This syndrome was called sudden arrhythmic death syndrome or sudden unexpected nocturnal death syndrome (SUNDS). Brugada syndrome was described as a cause for SUNDS seen in Thai men in 1997. [6] Brugada syndrome has been determined to be "phenotypically, genetically and functionally identical" to SUNDS in Thailand, where it is known as Lai Tai and Japan where it is known as Pokkuri and it has been proposed that there is similar equivalence between Brugada syndrome and Filipino subset of SUNDS, locally known as Bangungut ("nightmare"). [7] These cultures had been familiar with Brugada-linked SUNDS well before Occidental physicians had become acquainted with the pattern, with a Philippine medical journal noting "Such ‘deadly dreams’ are well known among the lay people, many of whom view them with sullen respect, if not frank terror." [8]"
This is all the Wikipedians interpretation of primary sources. Please use high quality secondary sources. Doc James ( talk · contribs · email) 22:32, 14 February 2019 (UTC)
References
{{
cite web}}
: Missing or empty |url=
(
help)
{{
cite web}}
: Unknown parameter |deadurl=
ignored (|url-status=
suggested) (
help)
{{
cite web}}
: Unknown parameter |deadurl=
ignored (|url-status=
suggested) (
help)
NORD2016
was invoked but never defined (see the
help page).{{
cite journal}}
: Check date values in: |date=
(
help)
{{
cite journal}}
: Text "date October 1997" ignored (
help)
Here are three secondary sources that cover the history of the condition
Please use these. Doc James ( talk · contribs · email) 22:55, 14 February 2019 (UTC)
What's wrong with this AHA 2018 review? [1] Yanping Nora Soong ( talk) 23:11, 14 February 2019 (UTC)
"Sudden unexplained nocturnal death syndrome, bangungut, pokkuri death syndrome [2]"
Why was this removed? Doc James ( talk · contribs · email) 22:33, 14 February 2019 (UTC)
See this 2018 review in JAHA: In the following decades, investigators have been engaged in revealing the relationship between SUNDS and BrS, but there still has not been a definitive conclusion.25, 32 Nevertheless, it is commonly recognized that VA is a major cause of SUNDS.32, 33 The following risk factors presumed to be involved in SUNDS may also trigger VA. Yanping Nora Soong ( talk) 23:28, 14 February 2019 (UTC)
References
GHR2015
was invoked but never defined (see the
help page).
![]() | Brugada syndrome has been listed as one of the
Natural sciences good articles under the
good article criteria. If you can improve it further,
please do so. If it no longer meets these criteria, you can
reassess it. Review: July 12, 2018. ( Reviewed version). |
![]() | Ideal sources for Wikipedia's health content are defined in the guideline
Wikipedia:Identifying reliable sources (medicine) and are typically
review articles. Here are links to possibly useful sources of information about Brugada syndrome.
|
![]() | A fact from Brugada syndrome appeared on Wikipedia's
Main Page in the
Did you know column on 17 August 2018 (
check views). The text of the entry was as follows:
| ![]() |
![]() | This article is rated GA-class on Wikipedia's
content assessment scale. It is of interest to the following WikiProjects: | |||||||||||||||||||||||||||||
|
There is a problem with the lower EKG image and its legend. The legend refers to two panels, one after treatment. The figure shows three panels for three different classes of the syndrome.
98.64.68.45 ( talk) 12:24, 8 October 2009 (UTC)
I agree with the poster below that the subjects should not be merged because their definitions differ. In addition, the disorders described as SUD (or SUNDS, Sudden Unexplained Nocturnal Death Syndrome) such as Bangungot can be classified as ethnomedical disorders. The description of the phenomena and traditional cures will undoubtedly differ in each case. To lump any or all of them together as Brugada seems reductionist and ethnocentric. Doing so would probably not only be scientifically wrong, but gives precedence to a biological cause and Western medical remedy (as found in the Brugada article) rather than allowing for a focus on the psycho-social reality of each disorder and valuable discussions of traditional remedies. I would suggest that there should be a section on linked ethnomedical disorders in the main Brugada article and that there should be links to each of the disorders' articles at the end of the Brugada article.
This link should take you to a 2002 article in Human Molecular Genetics which indicates a genetic link between SUNDS and Brugada: hmg.oxfordjournals.org/cgi/reprint/11/3/337.pdf
On a related note, More information on some of the ethnomedical disorders linked to SUNDS (particularly in Hmong men) can be found below. SUNDS has long been linked to sleep paralysis. See:
1) Hufford, David J. The Terror That Comes in the Night. Philadelphia: University of Pennsylvania Press, 1982. 2) Adler, Shelly R. "The Role of the Nightmare in Hmong Sudden Unexpected Nocturnal Death Syndrome: A Folkloristic Study of Belief and Health." Ph.D. diss., University of California--Los Angeles, 1991, 48-61. 3) Adler, Shelly R. "Sudden Unexpected Nocturnal Death Syndrome among Hmong Immigrants: Examining the Role of the 'Nightmare'," Journal of American Folklore 104 (1991): 54-71 Lorist 21:34, 6 August 2007 (UTC)
..one may call that an amateur page...
... and just what have _you_ written?
Brugada syndrome is a well defined entity, whereas the other ones are more ill-defined. There is no doubt that Brugada syndrome is a part of the others but other diseases is also part of SUDS and sudden death in asia. Therefore the subjects should not be merged.
A good reference:
1. Priori SG, Aliot E, Blomstrom-Lundqvist C, Bossaert L, Breithardt G, Brugada P, Camm AJ, Cappato R, Cobbe SM, Di Mario C, Maron BJ, McKenna WJ, Pedersen AK, Ravens U, Schwartz PJ, Trusz-Gluza M, Vardas P, Wellens HJ, Zipes DP. Task Force on Sudden Cardiac Death of the European Society of Cardiology. Eur Heart J. 2001 Aug;22(16):1374-450. ( Medline abstract)
Ksheka 11:54, Aug 24, 2004 (UTC)
Hi, I am an ICD patient myself as a result of going into ventricular fibrillation in my sleep in 2004. This is also known as SADS (Sudden arrhythmic death syndrome).
I have been genetically tested for the genes related to Brugada in 2005. The result was negative but I am having more genetic tests done. I have just recently had a blood sample taken for the second time. Hopefully they can research more into this. I have more information regarding brugada syndrome and my doctor has already met Dr. Brugada himself.
I would like to add more information about testing for Brugada. There is a drug called "ajmaline" which I was tested with to see if any abnormalities in my ECG showed up. Perhaps I could add this to the article and other information too? Smsmasters 09:02, 28 June 2007 (UTC)
All Class Ic "sodium current blocking agents" could be used to reveal hidden ECG abnormalities to diagnose this entity. However, prognostic significance of this test is quite questionable, indeed people without any VF episodes or spontaneous ECG changes has very little chance to develop a sudden cardiac death. Also, a new "old" drug is currently popular in treatment of Brugada syndrome: Quinidine. So I added some info about this topic with reference. —Preceding unsigned comment added by Lord Xar ( talk • contribs) 22:15, 18 September 2007 (UTC)
You could add a link to [www.brugadadrugs.org] on the wiki, a site discussing drugs associated with Brugada syndrome and lists of drugs to avoid.
Petertje puk ( talk) 19:16, 29 June 2009 (UTC)
Hello fellow Wikipedians,
I have just modified 3 external links on Brugada syndrome. Please take a moment to review my edit. If you have any questions, or need the bot to ignore the links, or the page altogether, please visit this simple FaQ for additional information. I made the following changes:
When you have finished reviewing my changes, please set the checked parameter below to true or failed to let others know (documentation at {{
Sourcecheck}}
).
This message was posted before February 2018.
After February 2018, "External links modified" talk page sections are no longer generated or monitored by InternetArchiveBot. No special action is required regarding these talk page notices, other than
regular verification using the archive tool instructions below. Editors
have permission to delete these "External links modified" talk page sections if they want to de-clutter talk pages, but see the
RfC before doing mass systematic removals. This message is updated dynamically through the template {{
source check}}
(last update: 5 June 2024).
Cheers.— InternetArchiveBot ( Report bug) 18:00, 9 November 2016 (UTC)
Hi @ Doc James:, I have just realised that the edits I made to the Brugada page yesterday inadvertently deleted the infobox, sorry.
I see that you in addition to restoring the infobox, you reverted the changes I made to the lead, with your comment being MEDMOS. I'd be grateful if you could help me understand in what way my version of the lead (pasted below) falls foul of MEDMOS - I felt it was an improvement as it avoided technical terms like 'vagal tone' which may be challenging for a lay reader to understand, while summarising the key points in the main body of the article below. I appreciate that you have a lot of Wiki experience and welcome your feedback. Thanks PeaBrainC ( talk) 21:49, 17 January 2018 (UTC)
@ Doc James:, PeaBrainC ( talk) 20:07, 18 January 2018 (UTC)
Brugada syndrome (BrS) is a
genetic condition which results in abnormal electrical activity within the
heart, increasing the risk of
abnormal heart rhythms and
sudden cardiac death.
[1] These abnormal heart rhythms often occur at rest or after a heavy meal, and can be triggered by a
fever, excessive alcohol, or certain medications.
[1] The symptoms of Brugada syndrome include
fainting but those affected may not have any symptoms at all.
[2]
As an inherited condition, about a quarter of those affected by Brugada syndrome will have a relative who also has the condition. [1] It may be caused by mutations in the SCN5A gene responsible for the cardiac sodium channel, although mutations causing the condition have also been found in other genes.
Brugada syndrome is typically diagnosed using an electrocardiogram (ECG), although medications such as ajmaline may be required to produce the characteristic ECG pattern. [1] There is no cure for Brugada syndrome, but the condition may be treated using an implantable cardioverter defibrillator (ICD). [3] Medications to help control the abnormal heart rhythms include isoproterenol in those who are acutely unstable, and quinidine. [4] The family members of a patient with Brugada syndrome may require testing for the condition. [4]
Were does this ref mention after meals or during sleep?
Not seeing "while
quinidine may be used in the longer term" in
https://rarediseases.org/rare-diseases/brugada-syndrome/ Found the ref and added it to the sentence in question.
Doc James ( talk · contribs · email) 18:49, 9 March 2018 (UTC)
GA toolbox |
---|
Reviewing |
Reviewer: Natureium ( talk · contribs) 16:25, 11 July 2018 (UTC)
I've never done a GAN review before, so please let me know (politely) if I'm doing anything wrong.
GA review – see WP:WIAGA for criteria
Mechanism: heart racing -> rapid heart rate : Done
PeaBrainC (
talk)
17:19, 13 July 2018 (UTC)
Hi @ Natureium:, thanks for reviewing this article. It's only my 2nd GAN but to my inexperienced eyes looks like you've done a good job. I will get on with making the amendments you have suggested and mark them as done on this page as I go. It may take a few days - busy times in the real world! PeaBrainC ( talk) 09:48, 12 July 2018 (UTC)
Sentence 4 begins: The abnormal heart rhythm often occurs. The The implies that the reader is to know which abnormal hear rhythm we are talkin about. But no abnormal heart rhythm has been mentioned, I think. That is, I think that there are heart disorders that are not abnomal heart rhythms. Even if there aren't, we should not assume that all readers know this.--
Ettrig (
talk) 20:57, 12 July 2018 (UTC) Done
PeaBrainC (
talk)
17:06, 13 July 2018 (UTC)
Hi Yanping, I wonder if we could have a discussion on the talkpage about our differences of opinion regarding the best form of language to use in the article. I don't want to edit war and am loathe to change your edits again without consensus, so I'm bringing it to the table here.
In your recent edit summaries you have suggested that "this page should be written for academics and professionals, if laypeople don’t know what sodium channels are they’re supposed to follow wikilinks" and that "this is a science article, not a brochure for patients". All the manuals of style suggest that articles should be written in a style suitable for a non specialist. From WP:BATTLE - "Texts should be written for everyday readers, not just for academics", and "While wikilinks should be provided for advanced terms and concepts in that field, articles should be written on the assumption that the reader will not or cannot follow these links, instead attempting to infer their meaning from the text."
In addition to our differences of opinion on writing style there are questions to be raised regarding the accuracy of what you have changed. Whether the prose is more or less technically oriented, it needs to be correct. For example, SCN5A is a gene. NaV1.5 is the protein forming the alpha subunit of the cardiac sodium channel that SCN5A encodes. INa is the current that NaV1.5 carries. These terms should not be mixed up as was done in your recent set of edits. Furthermore, Brugada has nothing to do with the late sodium current - increased late INa is responsible for the LQT3 form of long QT syndrome. Brugada is an oligogenic disorder but the association with sodium is entirely to do with a reduction in the peak current. It is wrong to say that BrS is due to mutations in SCN5A - a proportion of cases of BrS are associated with SCN5A mutations but in the majority no causative gene can be found.
Comparing your most recent version of the first paragraph of the causes section:
"The individual cells of the heart communicate with each other with electrical signals, and these electrical signals are disrupted in those with Brugada syndrome due to mutations in SCN5A, a cardiac sodium channel which is responsible for depolarizing the cardiomyocyte during phase 0 of the cardiac action potential by permitting the early component of the sodium current as well as permitting a residual current lasting throughout the low-conductance phases of repolarization, known as the late sodium current. Many of the genetic mutations that have subsequently been described in association with Brugada syndrome influence the early and late components of the sodium current in some way, often differentially, or affect other ionic currents."
to the previous version:
"The individual cells of the heart communicate with each other with electrical signals, and these electrical signals are disrupted in those with Brugada syndrome. As a genetic condition, the syndrome is ultimately caused by changes to a person's DNA, known as genetic mutations. The first mutations described in association with Brugada syndrome were in a gene responsible for a protein or ion channel that controls the flow of sodium ions through the cell membrane of heart muscle cells – the cardiac sodium channel. Many of the genetic mutations that have subsequently been described in association with Brugada syndrome influence the sodium current in some way, or affect other ionic currents."
I feel that the earlier version was more accurate and worded in a way more appropriate to a non-specialist audience. Like I say, I'm not going to revert again, but would appreciate the opinions of others who have previously shown an interest in the article. @ Yanping Nora Soong: @ Doc James: @ TylerDurden8823:. Thanks, PeaBrainC ( talk) 17:00, 14 February 2019 (UTC)
"As early as December 1981, the Center for Disease Control described sudden cardiac death during sleep of predominantly male Southeast Asian refugees. [5] According to the CDC report, "The abruptness of the deaths reported here is compatible with a cardiac dysrhythmia ... but the underlying mechanism remains unclear." This syndrome was called sudden arrhythmic death syndrome or sudden unexpected nocturnal death syndrome (SUNDS). Brugada syndrome was described as a cause for SUNDS seen in Thai men in 1997. [6] Brugada syndrome has been determined to be "phenotypically, genetically and functionally identical" to SUNDS in Thailand, where it is known as Lai Tai and Japan where it is known as Pokkuri and it has been proposed that there is similar equivalence between Brugada syndrome and Filipino subset of SUNDS, locally known as Bangungut ("nightmare"). [7] These cultures had been familiar with Brugada-linked SUNDS well before Occidental physicians had become acquainted with the pattern, with a Philippine medical journal noting "Such ‘deadly dreams’ are well known among the lay people, many of whom view them with sullen respect, if not frank terror." [8]"
This is all the Wikipedians interpretation of primary sources. Please use high quality secondary sources. Doc James ( talk · contribs · email) 22:32, 14 February 2019 (UTC)
References
{{
cite web}}
: Missing or empty |url=
(
help)
{{
cite web}}
: Unknown parameter |deadurl=
ignored (|url-status=
suggested) (
help)
{{
cite web}}
: Unknown parameter |deadurl=
ignored (|url-status=
suggested) (
help)
NORD2016
was invoked but never defined (see the
help page).{{
cite journal}}
: Check date values in: |date=
(
help)
{{
cite journal}}
: Text "date October 1997" ignored (
help)
Here are three secondary sources that cover the history of the condition
Please use these. Doc James ( talk · contribs · email) 22:55, 14 February 2019 (UTC)
What's wrong with this AHA 2018 review? [1] Yanping Nora Soong ( talk) 23:11, 14 February 2019 (UTC)
"Sudden unexplained nocturnal death syndrome, bangungut, pokkuri death syndrome [2]"
Why was this removed? Doc James ( talk · contribs · email) 22:33, 14 February 2019 (UTC)
See this 2018 review in JAHA: In the following decades, investigators have been engaged in revealing the relationship between SUNDS and BrS, but there still has not been a definitive conclusion.25, 32 Nevertheless, it is commonly recognized that VA is a major cause of SUNDS.32, 33 The following risk factors presumed to be involved in SUNDS may also trigger VA. Yanping Nora Soong ( talk) 23:28, 14 February 2019 (UTC)
References
GHR2015
was invoked but never defined (see the
help page).