AF4/FMR2 family member 1 is a
protein that in
humans is encoded by the AFF1gene.[5][6][7][8] At its same location was a record for a separate PBM1 gene, which has since been withdrawn and considered an alias. It was previously known as AF4 (ALL1-fused gene from chromosome 4).[8]
^Gu Y, Nakamura T, Alder H, Prasad R, Canaani O, Cimino G, Croce CM, Canaani E (November 1992). "The t(4;11) chromosome translocation of human acute leukemias fuses the ALL-1 gene, related to Drosophila trithorax, to the AF-4 gene". Cell. 71 (4): 701–8.
doi:
10.1016/0092-8674(92)90603-A.
PMID1423625.
S2CID6257922.
^Chen CS, Hilden JM, Frestedt J, Domer PH, Moore R, Korsmeyer SJ, Kersey JH (August 1993). "The chromosome 4q21 gene (AF-4/FEL) is widely expressed in normal tissues and shows breakpoint diversity in t(4;11)(q21;q23) acute leukemia". Blood. 82 (4): 1080–5.
doi:10.1182/blood.V82.4.1080.bloodjournal8241080 (inactive 31 January 2024).
PMID8353274.{{
cite journal}}: CS1 maint: DOI inactive as of January 2024 (
link)
Frestedt JL, Hilden JM, Kersey JH (August 1996). "AF4/FEL, a gene involved in infant leukemia: sequence variations, gene structure, and possible homology with a genomic sequence on 5q31". DNA and Cell Biology. 15 (8): 669–78.
doi:
10.1089/dna.1996.15.669.
PMID8769569.
Bertrand FE, Spengeman JD, Shah N, LeBien TW (December 2003). "B-cell development in the presence of the MLL/AF4 oncoprotein proceeds in the absence of HOX A7 and HOX A9 expression". Leukemia. 17 (12): 2454–9.
doi:
10.1038/sj.leu.2403178.
PMID14562113.
S2CID10639425.
Caslini C, Serna A, Rossi V, Introna M, Biondi A (June 2004). "Modulation of cell cycle by graded expression of MLL-AF4 fusion oncoprotein". Leukemia. 18 (6): 1064–71.
doi:
10.1038/sj.leu.2403321.
PMID14990976.
S2CID19189264.
AF4/FMR2 family member 1 is a
protein that in
humans is encoded by the AFF1gene.[5][6][7][8] At its same location was a record for a separate PBM1 gene, which has since been withdrawn and considered an alias. It was previously known as AF4 (ALL1-fused gene from chromosome 4).[8]
^Gu Y, Nakamura T, Alder H, Prasad R, Canaani O, Cimino G, Croce CM, Canaani E (November 1992). "The t(4;11) chromosome translocation of human acute leukemias fuses the ALL-1 gene, related to Drosophila trithorax, to the AF-4 gene". Cell. 71 (4): 701–8.
doi:
10.1016/0092-8674(92)90603-A.
PMID1423625.
S2CID6257922.
^Chen CS, Hilden JM, Frestedt J, Domer PH, Moore R, Korsmeyer SJ, Kersey JH (August 1993). "The chromosome 4q21 gene (AF-4/FEL) is widely expressed in normal tissues and shows breakpoint diversity in t(4;11)(q21;q23) acute leukemia". Blood. 82 (4): 1080–5.
doi:10.1182/blood.V82.4.1080.bloodjournal8241080 (inactive 31 January 2024).
PMID8353274.{{
cite journal}}: CS1 maint: DOI inactive as of January 2024 (
link)
Frestedt JL, Hilden JM, Kersey JH (August 1996). "AF4/FEL, a gene involved in infant leukemia: sequence variations, gene structure, and possible homology with a genomic sequence on 5q31". DNA and Cell Biology. 15 (8): 669–78.
doi:
10.1089/dna.1996.15.669.
PMID8769569.
Bertrand FE, Spengeman JD, Shah N, LeBien TW (December 2003). "B-cell development in the presence of the MLL/AF4 oncoprotein proceeds in the absence of HOX A7 and HOX A9 expression". Leukemia. 17 (12): 2454–9.
doi:
10.1038/sj.leu.2403178.
PMID14562113.
S2CID10639425.
Caslini C, Serna A, Rossi V, Introna M, Biondi A (June 2004). "Modulation of cell cycle by graded expression of MLL-AF4 fusion oncoprotein". Leukemia. 18 (6): 1064–71.
doi:
10.1038/sj.leu.2403321.
PMID14990976.
S2CID19189264.